|1.||D'Souza, A: 1 article (03/2011)|
|2.||Bastaki, S M A: 1 article (03/2011)|
|3.||Singh, J: 1 article (03/2011)|
|4.||Adem, A: 1 article (03/2011)|
|5.||Chandranath, S I: 1 article (03/2011)|
|1.||Stomach Ulcer (Gastric Ulcer)
03/01/2011 - "The results show that both PG and nitric oxide pathways can influence the inhibitory effect of PD-136450 against CRS-induced gastric ulcer. "
03/01/2011 - "PD-136450, an effective anti-secretory agent against gastrin- but not dimaprit-induced stimulation, evoked a dose-dependent inhibition of CRS-induced gastric ulcers. "
03/01/2011 - "The results of this study have demonstrated that although lansoprazole, PD-136450 and ranitidine were protective against CRS-induced gastric ulcers, the antiulcer activities of PD-136450 and ranitidine involved both PG and nitric oxide pathways, while lansoprazole acted independently of these two systems during CRS."
03/01/2011 - "The results showed that ranitidine was more potent than lansoprazole and PD-136450 in inhibiting CRS-induced gastric ulcers and its effect was shown to be influenced by PG as well as nitric oxide synthase. "
03/01/2011 - "This study compares the antiulcer activities of lansoprazole (a proton pump inhibitor), PD-136450 (CCK(2)/gastrin receptor antagonist) and ranitidine (histamine H(2) receptor antagonist) on CRS-induced gastric ulcers in rats. "
|2.||Nitric Oxide (Nitrogen Monoxide)
|4.||Cholecystokinin B Receptor
|5.||Proton Pumps (Proton Pump)
|6.||Histamine (Histamine Dihydrochloride)
|7.||Nitric Oxide Synthase (NO Synthase)