|1.||Otto, Michael: 3 articles (08/2014 - 09/2011)|
|2.||Kretschmer, Dorothee: 2 articles (08/2014 - 01/2012)|
|3.||Peschel, Andreas: 2 articles (08/2014 - 01/2012)|
|4.||Cheung, Gordon Y C: 1 article (08/2014)|
|5.||Kreiswirth, Barry N: 1 article (08/2014)|
|6.||Duong, Anthony C: 1 article (08/2014)|
|7.||Ho, Trung V: 1 article (08/2014)|
|8.||Chen, Yan: 1 article (08/2014)|
|9.||Yeh, Anthony J: 1 article (08/2014)|
|10.||Joo, Hwang-Soo: 1 article (08/2014)|
09/15/2012 - "When adjusted for total IgG levels and stratified for the presence of toxin in the infecting isolate as appropriate, the risk of sepsis was significantly lower in those patients with higher levels of IgG against α-hemolysin (Hla), δ-hemolysin (Hld), Panton Valentine leukocidin (PVL), staphylococcal enterotoxin C-1 (SEC-1), and phenol-soluble modulin α3 (PSM-α3). "
07/01/2005 - "Cell wall-associated and secreted proteins (e.g., protein A, hemolysins, and phenol-soluble modulin) and cell wall components (e.g., peptidoglycan and alanylated lipoteichoic acid) have been shown to be inflammatory, and these staphylococcal components may contribute to sepsis. "
02/01/2003 - "IECs were unresponsive to TLR2 ligands including the staphylococcal-derived Ags phenol soluble modulin, peptidoglycan, and lipotechoic acid and the mycobacterial-derived Ag soluble tuberculosis factor. "
07/15/2001 - "Here we report that HMEC are unresponsive to several additional biologically relevant TLR2 ligands, including, phenol-soluble modulin (PSM), a complex of three small secreted polypeptides from the skin commensal Staphylococcus epidermidis, soluble tuberculosis factor (STF), and Borrelia burgdorferi outer surface protein A lipoprotein (OspA-L). "
08/01/2014 - "Production of an attenuated phenol-soluble modulin variant unique to the MRSA clonal complex 30 increases severity of bloodstream infection."
09/01/2011 - "Staphylococcus aureus strains (n = 50) causing complicated skin and skin structure infections produced various levels of phenol-soluble modulin alpha-type (PSMα) peptides; some produced more than twice that produced by the control strain (LAC USA300). "
|4.||Soft Tissue Infections
09/15/2011 - "Here we developed a skin and soft tissue infection model in rabbits to test the hypothesis that PVL contributes to USA300 pathogenesis and compare it with well-established virulence determinants: alpha-hemolysin (Hla), phenol-soluble modulin-alpha peptides (PSMα), and accessory gene regulator (Agr). "
|5.||Formyl Peptide Receptors (Formyl Peptide Receptor)
|6.||GTP-Binding Proteins (G-Protein)
|7.||Proteins (Proteins, Gene)
|9.||Immunoglobulin G (IgG)
|10.||Hemolysin Proteins (Hemolysins)