|1.||Wang, Jian: 3 articles (10/2013 - 06/2005)|
|2.||Shimoda, Larissa A: 2 articles (07/2012 - 06/2005)|
|3.||Sylvester, J T: 2 articles (07/2012 - 06/2005)|
|4.||Gupte, Sachin A: 2 articles (06/2006 - 11/2004)|
|5.||Wolin, Michael S: 2 articles (06/2006 - 11/2004)|
|6.||Hong, S J: 2 articles (03/2005 - 12/2003)|
|7.||Liang, Hsiu-Chuan: 2 articles (03/2005 - 12/2003)|
|8.||Shen, Ching-Jung: 2 articles (03/2005 - 12/2003)|
|9.||Lin, Cheng-I: 2 articles (09/2004 - 04/2003)|
|10.||Chávez-Munguía, Bibiana: 1 article (12/2015)|
05/01/1994 - "Cyclopiazonic acid treatment during ischemia and reperfusion likewise decreased the incidence of ischemic and reperfusion arrhythmias. "
09/01/1995 - "Thapsigargin or cyclopiazonic acid proportionally depressed Ca2+ uptake rate +RY and -RY and produced distinctly different effects of ischemia. "
09/01/1995 - "Ischemia depressed Ca2+ uptake rate -RY at free [Ca2+] of 0.4 microM and above, while the depression + RY was significant only above 10 microM Ca2+. We tested the hypothesis that inhibition of the Ca-ATPase alone, by adding thapsigargin or cyclopiazonic acid, could reproduce the effects of ischemia on the homogenate Ca2+ uptake rate. "
01/01/2008 - "The effects of the SERCA inhibitors thapsigargin (10 nM-10 microM) and cyclopiazonic acid (CPA; 10-50 microM) and of the PMCA blockers eosin (1.5-10 microM) and o-vanadate (1-5 mM) on inner and outer hair cells (IHCs/OHCs) were examined in normoxia and ischemia using an in vitro model of the newborn rat cochlea. "
12/01/2003 - "Dependence of cyclopiazonic-acid-induced muscle contractures on extracellular Ca2+."
04/01/2003 - "Cyclopiazonic acid markedly depressed the relationship and increased the diastolic force (contracture) at high driving frequency, but did not induce action potentials during the rest interval. "
03/01/2005 - "Alteration of cyclopiazonic acid-mediated contracture of mouse diaphragm after denervation."
12/25/2009 - "Poisoning with the SR Ca(2+) pump blocker cyclopiazonic acid elicited a contracture of the fiber at the level of the overexpression site in presence and absence of external Ca(2+) which was not observed in control cells. "
07/01/1999 - "In the presence of cyclopiazonic acid 0.5 microM, the relationship between the amplitude of potassium contractures and the membrane potential shifted to more negative potentials, whereas the steady-state inactivation curve was unchanged. "
12/01/2003 - "Hypoxia also increased basal [Ca2+]cyt, the amount of CCE produced by store depletion with cyclopiazonic acid, and the amplitude of SOC-mediated currents (ISOC). "
06/01/2008 - "The ability to proliferate, the resting [Ca2+]cyt, and cyclopiazonic acid-induced capacitative Ca2+ entry in human PASMCs were enhanced significantly by chronic hypoxia compared with the control, and these effects were inhibited in a dose-dependent manner by capsazepine, a TRPV1 channel inhibitor. "
08/01/2009 - "The increase in frequency of slow waves by hypoxia was prevented by cyclopiazonic acid (CPA) but not by carbonyl cyanide m-chlorophenyl-hydrazone (CCCP), potassium cyanide (KCN) or low-Ca solution. "
06/01/2005 - "In PASMCs perfused with Ca2+-free Krebs Ringer bicarbonate solution (KRBS) containing cyclopiazonic acid to deplete Ca2+ stores in sarcoplasmic reticulum and nifedipine to prevent Ca2+ entry through L-type voltage-operated Ca2+ channels (VOCCs), hypoxia markedly enhanced both the increase in [Ca2+]i caused by restoration of extracellular [Ca2+] and the rate at which extracellular Mn2+ quenched fura-2 fluorescence. "
02/01/2002 - "3. The effect of hypoxia was completely prevented by thapsigargin or cyclopiazonic acid, selective inhibitors of sarcoplasmic reticulum Ca(2+) ATPase (SERCA). "
|1.||Ion Channels (Ion Channel)
|3.||Adenosine Triphosphatases (ATPase)
|4.||Caffeine (No Doz)
|7.||Sarcoplasmic Reticulum Calcium-Transporting ATPases
|8.||Fura-2 (Fura 2)