|1.||Richardson, Rudy J: 5 articles (02/2015 - 03/2008)|
|2.||Fink, John K: 5 articles (02/2015 - 03/2008)|
|3.||Glynn, Paul: 5 articles (03/2013 - 09/2005)|
|4.||Moretto, A: 4 articles (03/2007 - 02/2000)|
|5.||Chang, Ping-An: 4 articles (10/2006 - 04/2005)|
|6.||Wu, Yi-Jun: 4 articles (10/2006 - 04/2005)|
|7.||Hein, Nichole D: 3 articles (02/2015 - 07/2010)|
|8.||Vilanova, Eugenio: 3 articles (10/2014 - 04/2010)|
|9.||Lotti, M: 3 articles (03/2007 - 02/2000)|
|10.||dos Santos, Antonio Cardozo: 2 articles (08/2015 - 04/2015)|
08/01/2001 - "Certain esterase inhibitors protect from organophosphate-induced delayed polyneuropathy (OPIDP) when given before a neuropathic organophosphate by inhibiting neuropathy target esterase (NTE). "
03/15/2000 - "The target of promotion is unknown but there are indications that it might be similar and/or linked to neuropathy target esterase (NTE), which is the molecular target of organophosphate-induced delayed polyneuropathy (OPIDP). "
12/12/1997 - "Neuropathy target esterase (NTE) is suggested to be the molecular target for the initiation of the organophosphorus induced delayed polyneuropathy (OPIDP). "
06/01/1997 - "Organophosphate-induced delayed polyneuropathy (OPIDP) has been tested mainly byin vivo methods in the chicken and by examination of the inhibition of neuropathy target esterase in their neuronal tissue. "
12/01/1995 - "The identification of neuropathy target esterase (NTE) as the site for initiation of organophosphorus-induced delayed polyneuropathy (OPIDP) has led to informative acute and chronic neurotoxicity tests (adopted by OECD and EPA), to structure/activity and in vitro/in vivo predictions, and to a sound basis for extrapolations to man. "
11/18/2005 - "Reduction of neuropathy target esterase does not affect neuronal differentiation, but moderate expression induces neuronal differentiation in human neuroblastoma (SK-N-SH) cell line."
04/01/2005 - "Inhibition of neuropathy target esterase expressing by antisense RNA does not affect neural differentiation in human neuroblastoma (SK-N-SH) cell line."
01/01/1994 - "Neuropathy target esterase inhibition by organophosphorus esters in human neuroblastoma cells."
07/01/1993 - "Modification of mipafox-induced inhibition of neuropathy target esterase in neuroblastoma cells of human origin."
10/01/2006 - "In addition, activities of neuropathy target esterase and acetylcholinesterase were significantly reduced after exposure to 5 mM TOCP for 12 hr. Together, these results suggested that the loss of cytoskeletal components is the early event during the process of TOCP toxicity towards human neuroblastoma SK-N-SH cells."
01/01/2001 - "Determinations of phenyl-valerate hydrolase activity to assess the severity of an acute organophosphorous compounds poisoning cannot be recommended, but phenyl-valerate hydrolase may have utility in worker surveillance."
11/01/1981 - "In tissue from a fatal human poisoning and from hens given 4-8 x unprotected LD50 AChE was highly inhibited and neurotoxic esterase uninhibited. "
07/01/2010 - "Poisoning includes acute cholinergic crisis as a result of AChE inhibition, intermediate syndrome (IMS) due to neuromuscular necrosis and organophosphate-induced delayed neuropathy (OPIDN) due to inhibition of neuropathy target esterase (NTE). "
08/01/1999 - "Neuropathy target esterase (NTE), the putative target enzyme for organophosphate induced delayed polyneuropathy (OPIDP), can be measured in lymphocytes but has rarely been assessed in acute human poisoning. "
09/01/1993 - "Quinalphos was slowly absorbed from the gastrointestinal tract, as indicated by the severity of the cholinergic symptoms and the inhibition of neuropathy target esterase, which reached its maximum 72 and 96 h after poisoning. "
07/01/1990 - "Indices of organophosphorus (OP)-induced delayed neuropathy (OPIDN) in the hen model have traditionally been restricted to the early inhibition of neuropathy target esterase (NTE) and ataxia with associated pathological changes in hind limb peripheral nerve which occur more than 7 days after OP exposure. "
04/01/1993 - "Subsequent experiments in adult hens (the currently accepted animal model of choice for studies of OPIDN) showed that doses of CPS in excess of the LD50 in atropine-treated animals inhibited brain neurotoxic esterase (NTE) and produced mild to moderate ataxia. "
11/01/2001 - "The delayed neurotoxic potential of dichlorvos was assessed in terms of neuropathy target esterase (NTE) inhibition in the brain and the subsequent development of motor incoordination at 21 days post-exposure. "
06/01/1993 - "Relationship of neuropathy target esterase inhibition to neuropathology and ataxia in hens given organophosphorus esters."
06/01/2009 - "Neurotoxicity of phenyl saligenin phosphate (PSP) and lasalocid and possible interaction were studied in chickens by evaluating motor nerve conduction velocity (MNCV), clinical ataxia, and neuropathy target esterase (NTE) enzyme activity. "
09/15/1998 - "This study compares two direct-acting neuropathy target esterase (NTE) inhibitors (mipafox and 2-octyl-4H-1,3,2-benzodioxophosphorin 2-oxide (OBDPO)), a metabolic precursor to an NTE inhibitor (tri-o-cresyl phosphate or TOCP) and a potent acetylcholinesterase inhibitor (chlorpyrifos oxon or CPO) for their effects on outgrowth of neurite-like and cell processes and on viability in differentiated cultured cells (rat adrenal pheochromocytoma (PC-12) and brain glial tumor (C6)). "
|7.||tri-o-cresyl phosphate (TOTP)