|1.||Tsutsui, Takeki: 2 articles (02/2006 - 09/2005)|
|2.||Gupta, K: 1 article (05/2012)|
|3.||Hallikeremath, S R: 1 article (05/2012)|
|4.||Kale, A D: 1 article (05/2012)|
|5.||Kotrashetti, V S: 1 article (05/2012)|
|6.||Hauser, V: 1 article (08/2007)|
|7.||Frentzen, M: 1 article (08/2007)|
|8.||Braun, A: 1 article (08/2007)|
|9.||Hagiwara, Makoto: 1 article (02/2006)|
|10.||Watanabe, Eiko: 1 article (02/2006)|
12/01/1996 - "Neuronal cells were transiently stained with acid fuchsin, with a peak occurring at 24 h after the seizures. "
01/14/1994 - "Acid fuchsin may be a useful tool for delineating the distribution of reversibly injured immature neurons in experimental seizure paradigms."
10/01/1958 - "Acid fuchsin convulsions and electroshock in the mouse."
02/09/1995 - "To test this hypothesis in relation to seizures, bicuculline was given to rats in two sessions, separated by 1, 3, 5 or 7 days; seizure activity was recorded, and HSP-like immunoreactivity and neuronal injury (acid-fuchsin staining) were quantified in the CA3c sector of the hippocampus. "
10/17/1994 - "Intranigral injection of AP-7 increased the duration of electrographic seizure discharges and the number of HSP-positive and acid fuchsin stained cells in all hippocampal sectors, suggesting that blockade of the NMDA receptors in SNpr enhanced neural injury."
12/28/1995 - "These findings confirm that global ischemia injures microglia and astrocytes, and that cells in a given ischemic region sustain varying degrees of injury--from the HSP70 stained neurons that likely survive the ischemia to acid fuchsin stained cells that die."
04/01/1996 - "Forebrain ischemia was induced by bilateral carotid artery occlusion combined with systemic hypotension for 10 min. Cerebral perfusion-fixation was performed 7 days after ischemia, subsequent to which the brains were sectioned coronally and stained with celestine blue/acid fuchsin. "
03/01/2003 - "Whole brain histological evaluation was performed, and the sections were stained with hematoxylin and eosin (H and E), TUNEL, and vanadium acid fuchsin (VAF) staining to evaluate tissue effects, including apoptosis and ischemia. "
|5.||Status Epilepticus (Complex Partial Status Epilepticus)
01/14/1994 - "Subsequent to KA-induced status epilepticus in 11-day-old rats, acid fuchsin stain was evident in the hippocampal CA3, CA1, dentate gyrus and hilus by 24 h, peaked at 48 h and disappeared by 6 days, without evidence for neuronal loss. "
02/07/1997 - "We investigated this issue by inducing status epilepticus in normoxic (PaO2 approximately 100 mmHg) and hypoxia (PaO2 approximately 50 mmHg) rats, using heat-shock protein (HSP) expression as an index of early cell injury and acid fuchsin staining to detect cell death. "
|2.||Eosine Yellowish-(YS) (Eosin)
|3.||Methylene Blue (Methylthioninium Chloride)
|4.||Heat-Shock Proteins (Heat-Shock Protein)
|5.||Kainic Acid (Kainate)
|8.||Coloring Agents (Dyes)