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Hemodynamics during experimental gastric dilatation-volvulus in dogs.

Abstract
Gastric dilatation-volvulus (at 30 mm of Hg of gastric pressure) was experimentally induced in 8 dogs under sodium pentobarbital anesthesia. Hemodynamic indices including cardiac output, mean aortic pressure, left ventricular end diastolic pressure, heart rate, total peripheral resistance, and dp/dtmax were measured during a 180-minute period of gastric dilatation-volvulus and for 120 minutes after gastric decompression. Experimental gastric dilatation-volvulus resulted in significant (P less than 0.05) decreases in cardiac output (64%), mean aortic pressure (48%), left ventricular end diastolic pressure (68%), and ventricular dp/dtmax (32%) compared to 4 control dogs. Hemodynamic indices returned toward control values after gastric decompression. It was concluded that gastric dilatation-volvulus in the present experiment was capable of inducing hemodynamic alterations of a magnitude similar to those reported in gastric dilatation studies, using higher gastric pressures.
AuthorsE C Orton, W W Muir 3rd
JournalAmerican journal of veterinary research (Am J Vet Res) Vol. 44 Issue 8 Pg. 1512-5 (Aug 1983) ISSN: 0002-9645 [Print] United States
PMID6625301 (Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
Topics
  • Animals
  • Decompression (veterinary)
  • Dog Diseases (physiopathology)
  • Dogs
  • Female
  • Gastric Dilatation (complications, physiopathology, veterinary)
  • Hemodynamics
  • Ischemia (complications, veterinary)
  • Male
  • Myocardial Contraction
  • Pancreas (blood supply)
  • Pressure
  • Stomach (blood supply)
  • Stomach Volvulus (complications, physiopathology, veterinary)

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