Abstract | BACKGROUND: METHODOLOGY: PRINCIPAL FINDINGS: CONCLUSIONS: In addition to providing a rationale for the development of new approaches for the diagnosis and therapy of CVD, our observations may contribute to the elucidation of how anti- ApoA-I autoantibodies are elicited in individuals without autoimmune disease.
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Authors | Sabrina Pagano, Hubert Gaertner, Fabrice Cerini, Tiphaine Mannic, Nathalie Satta, Priscila Camillo Teixeira, Paul Cutler, François Mach, Nicolas Vuilleumier, Oliver Hartley |
Journal | PloS one
(PLoS One)
Vol. 10
Issue 7
Pg. e0132780
( 2015)
ISSN: 1932-6203 [Electronic] United States |
PMID | 26177543
(Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
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Chemical References |
- APOA1 protein, human
- Apolipoprotein A-I
- Autoantibodies
- Immobilized Proteins
- Immunoglobulin G
- Inflammation Mediators
- Interleukin-6
- Peptides
- Tumor Necrosis Factor-alpha
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Topics |
- Amino Acid Sequence
- Apolipoprotein A-I
(chemistry, immunology)
- Autoantibodies
(immunology)
- Cardiovascular Diseases
(blood, diagnosis, therapy)
- Circular Dichroism
- Humans
- Immobilized Proteins
(metabolism)
- Immunoglobulin G
(metabolism)
- Inflammation Mediators
(metabolism)
- Interleukin-6
(pharmacology)
- Molecular Sequence Data
- Peptides
(chemistry)
- Protein Engineering
- Protein Structure, Secondary
- Tumor Necrosis Factor-alpha
(pharmacology)
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