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Brugada syndrome and idiopathic left ventricular tachycardia unmasked by exercise and a class Ic drug.

Abstract
The patient was a 33-year-old male. Twenty years ago, he underwent radiofrequency catheter ablation for idiopathic sustained monomorphic ventricular tachycardia (VT) with an RBBB and superior axis pattern. The VT was inducible by programmed stimulation and entrained by rapid pacing. At this presentation, he developed palpitation and VT with the same morphology at the peak exercise on a treadmill with appearance of typical ECG pattern for Brugada syndrome (BrS). Pilsicainide induced the typical ECG pattern and premature ventricular beats (PVBs) of the same morphology as VT. The relationship between BrS and VT of left ventricular origin was discussed.
AuthorsSho Yuasa, Masahito Sato, Hitoshi Kitazawa, Masaaki Okabe, Yasushi Komatsu, Tomoyasu Koshikawa, Seiichi Miyajima, Yoshifusa Aizawa
JournalJournal of electrocardiology (J Electrocardiol) 2014 Sep-Oct Vol. 47 Issue 5 Pg. 721-4 ISSN: 1532-8430 [Electronic] United States
PMID24908473 (Publication Type: Case Reports, Journal Article)
CopyrightCopyright © 2014 Elsevier Inc. All rights reserved.
Chemical References
  • Anti-Arrhythmia Agents
  • Lidocaine
  • pilsicainide
Topics
  • Adult
  • Anti-Arrhythmia Agents
  • Brugada Syndrome (diagnosis, physiopathology, therapy)
  • Cardiac Catheterization
  • Defibrillators, Implantable
  • Electrocardiography
  • Exercise Test
  • Humans
  • Lidocaine (analogs & derivatives)
  • Male
  • Tachycardia, Ventricular (diagnosis, physiopathology, therapy)

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