Oculomotor neuromyotonia with lid ptosis on abduction.

Abstract

Ocular neuromyotonia is an episodic involuntary contraction of one or more of the extraocular muscles resulting from spontaneous neural discharges of the oculomotor nerve. It is characterized by symptomatic diplopia for the duration of the contraction and occurs either spontaneously or after a sustained eccentric gaze. Although the mechanism of ocular neuromyotonia is not fully understood, primary or secondary aberrant regeneration and consequent ephaptic transmission generally are held to be involved. We report the case of a patient with right oculomotor neuromyotonia accompanied by lid ptosis on abduction of the left eye who was successfully treated with oral carbamazepine.

• Authors: Si Bum Kim, Sei Yeul Oh, Moo Hwan Chang, Sungeun E Kyung
• Journal: Journal of AAPOS : the official publication of the American Association for Pediatric Ophthalmology and Strabismus (J AAPOS) Vol. 17 Issue 1 Pg. 97-9 (Feb 2013) ISSN: 1528-3933 [Electronic] United States
• PMID: 23337349 (Publication Type: Case Reports, Journal Article)
• Copyright: Copyright © 2013 American Association for Pediatric Ophthalmology and Strabismus. Published by Mosby, Inc. All rights reserved.

Chemical References

• Analgesics, Non-Narcotic
• Carbamazepine

Topics

• Analgesics, Non-Narcotic (therapeutic use)
• Blepharoptosis (drug therapy, etiology)
• Carbamazepine (therapeutic use)
• Diplopia (drug therapy, etiology)
• Humans
• Isaacs Syndrome (drug therapy, etiology)
• Male
• Middle Aged
• Ocular Motility Disorders (complications, drug therapy)
• Oculomotor Muscles (innervation)
• Oculomotor Nerve Diseases (drug therapy, etiology)
• Visual Acuity