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Presentation of opsoclonus myoclonus ataxia syndrome with glutamic acid decarboxylase antibodies.

Abstract
In this rare case, the patient presented with opsoclonus, myoclonus and ataxia. Serological and imaging studies revealed high glutamic acid decarboxylase antibody (GAD-Ab) levels. High-dose corticosteroids were of no benefit and subsequent intravenous immunoglobulin (IVIg) administration proved resolution of the condition. Levetiracetam proved useful in symptomatically controlling the myoclonus. Follow-up GAD-Ab levels were within normal limits.
AuthorsHanul Srinivas Bhandari
JournalBMJ case reports (BMJ Case Rep) Vol. 2012 (Aug 08 2012) ISSN: 1757-790X [Electronic] England
PMID22878989 (Publication Type: Case Reports, Journal Article, Review)
Chemical References
  • Anticonvulsants
  • Autoantibodies
  • Immunoglobulins, Intravenous
  • Levetiracetam
  • Glutamate Decarboxylase
  • Piracetam
Topics
  • Adult
  • Anticonvulsants (therapeutic use)
  • Anxiety (drug therapy)
  • Ataxia (immunology)
  • Autoantibodies (immunology)
  • Diagnosis, Differential
  • Earache (immunology)
  • Female
  • Follow-Up Studies
  • Glutamate Decarboxylase (immunology)
  • Headache (immunology)
  • Humans
  • Immunoglobulins, Intravenous (therapeutic use)
  • Lethargy (immunology)
  • Levetiracetam
  • Meningoencephalitis (diagnosis)
  • Opsoclonus-Myoclonus Syndrome (drug therapy, immunology, physiopathology)
  • Piracetam (analogs & derivatives, therapeutic use)
  • Treatment Outcome

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