Beta-galactosylceramide alters invariant natural killer T cell function and is effective treatment for lupus.
Abstract |
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Authors | Sufi R Morshed, Tsuyoshi Takahashi, Paul B Savage, Neeraja Kambham, Samuel Strober |
Journal | Clinical immunology (Orlando, Fla.)
(Clin Immunol)
Vol. 132
Issue 3
Pg. 321-33
(Sep 2009)
ISSN: 1521-7035 [Electronic] United States |
PMID | 19564135
(Publication Type: Journal Article)
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Chemical References |
- Antibodies, Antinuclear
- Antigens, CD1d
- Ceramides
- Monosaccharides
- Receptors, Antigen, T-Cell
- Receptors, Antigen, T-Cell, alpha-beta
- beta-galactosyl ceramide
- Interleukin-4
- Interferon-gamma
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Topics |
- Animals
- Antibodies, Antinuclear
(blood)
- Antigens, CD1d
(immunology, metabolism)
- B-Lymphocytes
(drug effects, immunology, metabolism)
- Ceramides
(immunology, metabolism, pharmacology, therapeutic use)
- Female
- Interferon-gamma
(blood)
- Interleukin-4
(blood)
- Kidney
(drug effects, pathology)
- Liver
(immunology)
- Lupus Erythematosus, Systemic
(immunology, pathology, therapy)
- Lupus Nephritis
(immunology, pathology, therapy)
- Mice
- Mice, Inbred C57BL
- Mice, Inbred NZB
- Monosaccharides
(immunology, metabolism, pharmacology, therapeutic use)
- Natural Killer T-Cells
(drug effects, immunology, metabolism)
- Proteinuria
(pathology, urine)
- Receptors, Antigen, T-Cell
(immunology, metabolism)
- Receptors, Antigen, T-Cell, alpha-beta
(immunology, metabolism)
- Spleen
(immunology)
- Survival Analysis
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