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Myomatous erythrocytosis syndrome.

Abstract
Uterine myomas are common but erythrocytosis caused by these is rarely seen. We report a case that illustrates the conjunction of various aetiological factors required for this clinical entity to evolve. A voluminous, retroperitoneally located and focally degenerated myoma was associated with severe secondary erythrocytosis (haematocrit: 65.5%) which resolved after hysterectomy. It has been demonstrated previously that myomatous tissue is the source of excessive production of erythropoietin. Local tissue hypoxia, which is more prone to develop in a pedunculated myoma, stimulates the process. Other prerequisites are a very large size of the myoma and the absence of menometrorrhagia of a severity such as to cause a depletion in iron reserves.
AuthorsChristophe Blockeel, Bart Op De Beeck, Claire Bourgain, Jean-Jacques Amy
JournalThe National medical journal of India (Natl Med J India) 2005 Sep-Oct Vol. 18 Issue 5 Pg. 247-9 ISSN: 0970-258X [Print] India
PMID16433138 (Publication Type: Case Reports, Journal Article)
Chemical References
  • Receptors, Erythropoietin
  • Erythropoietin
Topics
  • Adult
  • Erythropoietin (metabolism)
  • Female
  • Humans
  • Immunohistochemistry
  • Leiomyoma (diagnosis, diagnostic imaging)
  • Polycythemia (diagnosis, diagnostic imaging)
  • Receptors, Erythropoietin (metabolism)
  • Syndrome
  • Ultrasonography
  • Uterine Neoplasms (diagnosis, diagnostic imaging)

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