Polyuria

08/15/2008 - "Desmopressin is most effective in children with nocturnal polyuria and normal bladder capacity. "
09/01/1985 - "All patients had a significant reduction of their polyuria after a peroral dose of 50 micrograms DDAVP. "
11/01/2015 - "Desmopressin is an effective treatment for nocturnal polyuria in elderly women, where conservative treatment has failed. "
07/01/1999 - "Desmopressin is an effective treatment for nocturnal polyuria in some elderly men. "
07/01/1999 - "To assess whether desmopressin (1-desamino 8-d-arginine vasopressin) is safe and effective in the treatment of nocturnal polyuria in elderly men. "

03/01/1998 - "Moreover, it was effective in inducing the following during polyuria: (a) a depression of the diuretic response to water load in presence of a reduction in plasma osmolality; (b) a reduction in creatinine cl. "
01/01/1991 - "A polyuria and a decreased creatinine clearance are reported on day 8. We were interested in a special study of this polyuria. "
12/01/2015 - "The most common side effect of polyuria only weakly predicts increasing creatinine or reduced kidney function. "
01/01/2013 - "After 6 weeks of standard antituberculous treatment, progressive deterioration of his renal function was observed: creatinine levels rose from 38.9μmol/L to 318.2μmol/L and urine volume also progressively increased to reach a state of true polyuria (8 to 10L of urine per day). "
06/01/2011 - "Elimination of other common urinary solutes relative to creatinine was generally maintained during the polyuria phase, except that phosphate excretion increased temporarily. "

03/01/2006 - "Empiric treatment with vasopressin improved the polyuria. "
10/01/2009 - "To develop what we believe to be the first viable animal model of XNDI, we generated mice in which the V2R gene could be conditionally deleted during adulthood by administration of 4-OH-tamoxifen.Radioligand-binding studies confirmed the lack of V2R-binding sites in kidneys following 4-OH-tamoxifen treatment, and further analysis indicated that upon V2R deletion, adult mice displayed all characteristic symptoms of XNDI, including polyuria, polydipsia, and resistance to the antidiuretic actions of vasopressin. "
01/01/1997 - "Studies in Brattle Boro rats which are vasopressin deficient, revealed low levels of AQP2 in association with extreme polyuria. "
04/01/2015 - "Night polyuria was caused by the deficiency of vasopressin in most cases (37.6%) and occurred mainly (24.7%, group D) in a conjunction with small bladder capacity, and rarely alone (12.9%, group B). "
04/01/2015 - "Too small bladder capacity occurred mainly (37.6%, group C) as the only etiological factor of PMNE, and in 24.7% (group A) in a conjunction with nocturnal polyuria due to decreased excretion of vasopressin. "

10/01/2005 - "The present study confirms that AQP3 null mice exhibit severe polyuria and polydipsia and demonstrated that they exhibit increased urinary sodium excretion. "
10/01/2015 - "Polyuria often occurred in neurocritical care patients, but was not usually associated with sodium imbalance, CSW or CDI. "
10/01/2015 - "Polyuria was defined as diuresis above 4000 ml/day, hyponatraemia as a serum sodium (SeNa) < 135 mmol/l and hypernatraemia as SeNa > 150 mmol/l. "
10/01/2015 - "Polyuria has the potential to cause severe water and sodium imbalance. "
03/01/2012 - "EFWC was useful in the differential diagnosis of polyuria during rising serum sodium levels, while FWC was misleading."

02/01/2015 - "This study aimed to establish independent risk factors for polyuria in a community, secondary-level lithium-treated sample of patients. "
10/01/2013 - "The available evidence is contradictory as to whether SDD of lithium reduces polyuria; however, no trial has demonstrated any downfall of SDD in terms of prophylactic efficacy or adverse effects. "
06/01/2011 - "Distress due to lithium-induced polyuria: exploratory study."
03/01/1983 - "The mechanism of polyuria in rats pretreated with lithium studies by in vitro microperfusion."
08/01/2015 - "Symptomatic questioning, EMUO, and an FIR could be used in clinical practice to inform the prescriber of the probability that a lithium-treated patient is experiencing polyuria. "

07/01/1982 - "Prevention of polydipsia improved the polyuria but not the concentration defect seen 8 days after cisplatin. "
04/01/2013 - "The majority of cisplatin-treated patients develop hypomagnesaemia, often associated with a reduced glomerular filtration rate (GFR), polyuria and other electrolyte disturbances. "
01/01/1993 - "Mechanism of polyuria after cisplatin therapy."
04/01/1990 - "Five days after cisplatin, kidney function was significantly better than in rats treated with cisplatin alone (ClCr 0.21 +/- 0.06 vs. 0.03 +/- 0.01 ml min-1 x 100 g BW-1), after 28 days the only sign of nephrotoxicity was polyuria (UVol 28.9 +/- 3.7 vs. 19.0 +/- 2.6 ml 24 h-1) after 84 days no differences could be observed at all. "
01/01/1988 - "The long-lasting tubular injury and the slowness of repair are consistent with the chronic renal dysfunction (polyuria and hypomagnesemia) that cisplatin is known to induce in both man and experimental animals."

06/01/2014 - "Both children were treated with indomethacin resulting in improved linear growth and polyuria. "
02/01/2001 - "During polyuria significant positive correlations occurred between the absolute effects of indomethacin on 6KPGF excretion and those on functional excretory parameters. "
07/01/1990 - "By reducing urine output, indomethacin controlled both in utero and extrauterine polyuria in this patient."
12/01/1989 - "II. Indomethacin and effects of lysine-8-vasopressin in hypotonic polyuria]."
11/01/1989 - "Later an additional treatment with indomethacin (2 mg/kg/d) was necessary in 3 boys because of an increase of polyuria; there was a further reduction of the daily urinary volume of 50-60%. "

01/01/2012 - "Li-induced polyuria, and decreases in urine osmolality and AQP2 protein abundance in the renal medulla, were significantly less compared with WT mice despite the lack of differences in Li intake or terminal serum or inner medullary tissue Li levels. "
01/01/2010 - "Enhanced expression of renal aquaporin-2 (AQP2) has been reported when polyuria occurs in diabetic animal models. "
02/01/2007 - "The polyuria might also be attributed to reduced aquaporin 2 expression and the attendant urinary concentrating defect. "
05/31/2002 - "Severe polyuria was observed as an early event with no other obvious sequelae after Stx administration, in parallel with elevated urinary level of aquaporin 2 (AQP2) water channel protein that was determined by a sandwich EIA assay. "
06/01/1999 - "The present study was undertaken to determine whether urinary excretion of aquaporin-2 (AQP-2) water channel under ad libitum water intake is of value to differentiate polyuria caused by psychogenic polydipsia from central diabetes insipidus. "

01/01/2012 - "In the kidney, deletion of UT-A1/UT-A3 results in polyuria and a severe urine concentrating defect, indicating that intrarenal recycling of urea plays a crucial role in the overall capacity to concentrate urine. "
06/01/2010 - "The polyuria observed in homozygotes may be due to the combination of two additive factors, a decrease in activity of mutant NKCC2 and an increase in medullary blood flow, due to prostaglandin-induced vasodilation, that impairs countercurrent exchange of urea in the medulla. "
06/01/2010 - "Mutation of the Na(+)-K(+)-2Cl(-) cotransporter NKCC2 in mice is associated with severe polyuria and a urea-selective concentrating defect without hyperreninemia."
09/01/1991 - "A water deprivation test was conducted on three animals: polyuria disappeared and plasma urea increased significantly."
03/01/1998 - "Following transplantation, useful parameters were a high diuresis volume at time zero along with low urine NAG and high albumin outputs; early (first opst-graft 3 d) polyuria, low urea and GGT, high K, NAG and total protein excretions."

01/01/2015 - "The main differential diagnoses causing HN are the syndrome of inappropriate antidiuretic hormone secretion, marked by inappropriate retention of water, and cerebral salt wasting, characterized by polyuria and natriuresis. "
12/01/2014 - "Decades later, a subset of patients with polyuria unresponsive to therapy were recognized, leading to the distinction between central DI and nephrogenic DI, an early example of a hormone-resistant condition. "
12/01/2014 - "Early in the 20th century, the amelioration of the polyuria with extracts of the posterior pituitary inaugurated a new era in therapy and advanced the hypothesis that DI was due to a hormone deficiency. "
08/01/2008 - "Although the pathophysiology of nocturnal polyuria remains obscure, some investigators believe that low night-time levels of antidiuretic hormone are involved. "
02/01/2008 - "Increase in 24-hour urine production/weight causes nocturnal polyuria due to impaired function of antidiuretic hormone in elderly men."

09/01/2008 - "Observed NHE-I-dependent changes in electrolyte excretion, polyuria, and reduced protein reabsorption in the acute postoperative phase are predictors of favorable graft outcome in humans."
10/01/1998 - "Hypernatraemia and polyuria in a patient with acute myeloid leukaemia and allogeneic bone marrow transplant."
10/01/1997 - "Complaints of polyuria and polydipsia necessitated biopsies of the grafts to rule out graft rejection. "
09/01/1990 - "Irrespective of the route of venous drainage pancreas transplants normalized the diabetic polyuria, polyphagia, and polydipsia. "
01/01/2015 - "Polyuria after kidney transplantation causes graft dysfunction and increased thrombotic risk. "

10/01/1991 - "Our study documents the following: (1) only 2 of 103 (2%) patients referred for parathyroidectomy had "true" asymptomatic hyperparathyroidism; (2) only symptoms of fatigue, bone pain, and weight loss correlated with the degree of hypercalcemia, whereas muscular weakness, psychiatric symptoms, nocturia, polyuria, recent memory loss, constipation, and nephrolithiasis did not; (3) only 1 of 15 patients who were referred as asymptomatic were truly asymptomatic after more thorough questioning, and all 14 improved following parathyroidectomy; (4) 81% of the patients who were referred with symptoms improved following parathyroidectomy; and (5) permanent complications occurred in only 4 patients. "

06/01/2003 - "Polyuria and/or polydipsia, present in 11 horses, was reduced in all after treatment. "
04/01/1986 - "As a teenager she no longer had thirst or polyuria after treatment was stopped. "
07/01/1999 - "A 16-year-old boy presented with a four-month history of polyuria-polydipsia and a diplopia which had reverted after treatment. "
01/01/1991 - "It caused severe progressive proteinuria followed by polyuria and glucosuria, reaching maximum levels at 3 days after treatment in the SC group, whereas it produced mild proteinuria without glucosuria in the IP group. "
08/01/2008 - "Twenty-three days after treatment, polydipsia and polyuria were relieved, and acidosis, kaliopenia and anemia were improved in the patient. "

01/01/2009 - "After systemic chemotherapy, the patient developed tumor lysis syndrome followed by a phase of proximal tubule dysfunction presenting as polyuria and diverse electrolyte abnormalities. "
03/01/1995 - "The rarity of polyuria at presentation, the role of renal biopsy in the diagnosis, and the response of renal failure to specific chemotherapy are discussed."
11/01/1994 - "Hypopituitarism due to an intracranial tumor and the extracellular volume excess due to chemotherapy caused hyponatremia and polyuria. "
12/01/2008 - "Cancer patients who have symptoms such as nausea, vomiting, polyuria and polydipsia while they are not on chemotherapy should be evaluated for not only metabolic complications like hypercalcemia but also posterior pituitary or stalk metastasis MRI could be the choice of imaging for pituitary metastasis."
01/01/2006 - "We report the case of a 48-year-old woman suffering from diffuse large cell lymphoma, who developed hypotonic polyuria, hypernatriaemia and somnolence after the first course of chemotherapy with CHOEP and rituximab. "

03/28/2014 - "Oral administration of SAT did not exhibit toxicity and death at a dose not more than 2000 mg/kg. SAT dose-dependently improved the symptoms of polydipsia, polyuria, polyphagia and weight loss in diabetic rats. "
12/01/1992 - "While the oral administration caused saline polydipsia and polyuria typical of chronic systemic mineralocorticoid excess, the icv licorice derivatives produced hypertension without affecting saline appetite. "
07/01/1997 - "5. No polyuria or glucosuria was observed following the oral administration of ornidazole, suggesting that any (R)-3-chlorolactate produced was insufficient to affect renal metabolism. "
05/15/1979 - "Several factors may have contributed to the decrease in urine struvite concentration, including eradication of a urease-producing Proteus sp from the urinary tract and induction of polydipsia and compensatory polyuria by oral administration of sodium chloride."