Phosphates

06/01/1996 - "Our results suggest that MIA reduced reperfusion arrhythmias and improved functional recovery in isolated rat hearts subjected to global ischemia apparently by preserving high-energy phosphates during ischemia and by inhibiting Na+/H+ exchange, with attenuated cellular imbalance between Na+ and Ca2+."
01/01/1996 - "Hemodynamic results of a previous study with an identical protocol were reanalyzed; a biochemical analysis of myocardial high-energy phosphates was investigated after 60 min of global ischemia and at the end of the experiments after 45 min of reperfusion. "
11/01/1993 - "This study investigates the relationship between activation of KATP and preservation of high-energy phosphates during global no-flow ischemia in arterially perfused guinea pig right ventricular walls. "
08/01/1993 - "This study was focused on the changes of high energy phosphates and tissue blood flow (TBF) in ischemia and reperfusion, of jaundiced rat liver. "
02/01/1990 - "Previous studies have noted that myocardial blood flow and high energy phosphates are heterogeneous across the myocardial wall during ischemia. "

01/01/1984 - "In addition to confirming previously reported findings related to changes in high-energy phosphates consequent to anoxia, 31P nuclear magnetic resonance studies of rat brain indicated that maintenance of energy-rich phosphates is less effective in the aged brain than in the mature cerebrum."
07/01/2012 - "In this study, the microvascular tissue oxygenation and oxidative muscle metabolism during graded hypoxia and reoxygenation were examined in a rabbit model by near-infrared (NIR) spectroscopy and correlated with high-energy phosphates measured by (31)P nuclear magnetic resonance (NMR) spectroscopy. "
05/01/1995 - "This study investigates the relation between myocardial oxygen consumption (MVO2), function, and high energy phosphates during severe hypoxia and reoxygenation in sheep in vivo. "
01/01/1995 - "The effect of prolonged anoxia at 3 degrees C on tissue high energy phosphates and phosphodiesters in turtles: a 31P-NMR study."
04/01/1994 - "The results of this in vitro study suggest that relatively long periods of hypoxia lead to a deficiency of high energy phosphates in the cell. "

10/26/1990 - "Animal studies have provided the basis for understanding changes in high-energy phosphates with myocardial ischemia. "
11/01/2003 - "PCr restores high-energy phosphates and attenuates metabolic stress during periods of myocardial ischemia in the rat. "
04/01/1997 - "One aim of coronary reperfusion after myocardial ischemia is to restore the myocardial content of high energy phosphates. "
10/01/1994 - "Alteration of the cytosolic-mitochondrial distribution of high-energy phosphates during global myocardial ischemia may contribute to early contractile failure."
02/01/1992 - "During myocardial ischemia there is a drop in high-energy phosphates in the myocardium. "

02/01/1995 - "We conclude that the improved functional recovery in preconditioning is not due to a higher level of high energy phosphates or less acidosis during the final reperfusion."
12/01/1996 - "This study used in vivo 31P nuclear magnetic resonance spectroscopy to determine the relationship of intracellular tissue acidosis and delayed recovery of brain high-energy phosphates to increased damage during the reperfusion period. "
01/01/2008 - "Thus, it appears that not acidosis but depletion of high energy phosphates disabled muscle function and therefore may have been the cause of death of the non-survivors."
10/01/1995 - "This resulted in a better maintenance of high-energy phosphates and a lower acidosis as measured by 31P nuclear magnetic resonance spectroscopy. "
01/01/1995 - "Factors which influence alterations of phosphates and pH in exercising human skeletal muscle: measurement error, reproducibility, and effects of fasting, carbohydrate loading, and metabolic acidosis."

03/01/2000 - "Wavefront necrosis starts from the subendocardium, but the production of high-energy phosphates in the subepicardium is known to increase and compensate for the reduction in high-energy phosphate production in the subendocardium. "
11/01/1997 - "In conclusion, depletion of high-energy phosphates seems to occur as a result of pancreatic cell death rather than being a cause of pancreatic necrosis. "
09/01/1987 - "Rectal necrosis is a complication of injury during the administration of phosphates enemata which, although rare, deserves greater awareness. "
11/01/1986 - "The present study was designed to simulate the clinical situation of reocclusion and determine whether verapamil might be effective in reducing myocardial necrosis and preserving high energy phosphates in this setting. "
04/15/1999 - "We examined the phosphorylation state of transmembrane tumour necrosis factor-alpha (mTNF) with [32P]orthophosphate labelling and in vitro kinase assays, in lipopolysaccharide-stimulated RAW264.7 cells. "