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N-n-butyl haloperidol iodide

a cardioprotective agent; structure in first source

Networked: 20 relevant articles (6 outcomes, 5 trials/studies)

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Bio-Agent Context: Research Results

Organic Chemicals: 133
- Ketones: 2618
  - Butyrophenones: 43
    - Haloperidol: 5363
      - N-n-butyl haloperidol iodide: 20

Experts

1.	Shi, Ganggang: 11 articles (10/2021 - 01/2007)
2.	Zhang, Yanmei: 10 articles (01/2019 - 01/2007)
3.	Shi, Gang-Gang: 9 articles (01/2022 - 08/2003)
4.	Chen, Yicun: 9 articles (01/2019 - 01/2010)
5.	Gao, Fenfei: 9 articles (01/2019 - 01/2010)
6.	Wang, Bin: 8 articles (01/2022 - 04/2012)
7.	Zheng, Fuchun: 7 articles (01/2019 - 01/2013)
8.	Zhong, Shuping: 7 articles (01/2019 - 01/2013)
9.	Gao, Fen-Fei: 6 articles (01/2022 - 06/2004)
10.	Zhang, Yan-Mei: 6 articles (01/2022 - 06/2006)

Related Diseases

1.	Hypoxia (Hypoxemia) 06/07/2016 - "N-n-butyl haloperidol iodide ameliorates hypoxia/reoxygenation injury through modulating the LKB1/AMPK/ROS pathway in cardiac microvascular endothelial cells." 06/07/2016 - "We have reported that N-n-butyl haloperidol iodide (F2) can attenuate hypoxia/reoxygenation (H/R) injury in cardiac microvascular endothelial cells (CMECs). " 01/01/2016 - "N-n-butyl Haloperidol Iodide Protects against Hypoxia/Reoxygenation Injury in Cardiac Microvascular Endothelial Cells by Regulating the ROS/MAPK/Egr-1 Pathway." 09/22/2015 - "N-n-butyl haloperidol iodide protects cardiomyocytes against hypoxia/reoxygenation injury by inhibiting autophagy." 07/02/2015 - "Effect of N-n-butyl haloperidol iodide on ROS/JNK/Egr-1 signaling in H9c2 cells after hypoxia/reoxygenation."
2.	Myocardial Ischemia (Ischemic Heart Diseases) 01/01/2010 - "Our previous studies have shown that N-n-butyl haloperidol iodide (F2) can antagonize myocardial ischemia/reperfusion (I/R) injury by down-regulating the early growth response (Egr)-1 expression, but the molecular mechanisms are not well understood. " 01/01/2009 - "Our previous studies have shown that N-n-butyl haloperidol iodide (F(2)) can antagonize myocardial ischemia/reperfusion (I/R) injury by blocking intracellular Ca(2+) overload and suppressing Egr-1 overexpression. " 01/01/2007 - "Our previous studies have shown that N-n-butyl haloperidol iodide (F(2)) can antagonize myocardial ischemia/reperfusion (I/R) injury by blocking intracellular Ca(2+) overload. " 01/01/2014 - "N-n-butyl haloperidol iodide (F2), a novel compound, has shown palliative effects in myocardial ischemia/reperfusion (I/R) injury. " 01/01/2007 - "The protective effects of N-n-butyl haloperidol iodide on myocardial ischemia-reperfusion injury in rats by inhibiting Egr-1 overexpression."
3.	Reperfusion Injury 01/01/2007 - "The protective effects of N-n-butyl haloperidol iodide on myocardial ischemia-reperfusion injury in rats by inhibiting Egr-1 overexpression." 06/30/2004 - "Protective effects of N-n-butyl haloperidol iodide on myocardial ischemia-reperfusion injury in rabbits." 06/01/2006 - "Effects of N-n-butyl haloperidol iodide on myocardial ischemia/reperfusion injury and Egr-1 expression in rat." 01/01/2013 - "N-n-butyl haloperidol iodide (F2) has been shown to antagonize myocardial ischemia/reperfusion injury by blocking calcium channels. " 08/01/2003 - "Effects of N-n-butyl haloperidol iodide on rat myocardial ischemia and reperfusion injury and L-type calcium current."
4.	Liver Cirrhosis (Hepatic Cirrhosis) 01/01/2022 - "N-n-Butyl haloperidol iodide ameliorates liver fibrosis and hepatic stellate cell activation in mice."
5.	Ischemia 08/01/2003 - "To study the effects of N-n-butyl haloperidol iodide (F2) on rat heart ischemia/reperfusion (I/R) injury and L-type calcium current (ICa) in rat ventricular myocytes. " 09/22/2015 - "N-n-butyl haloperidol iodide (F2), a novel compound derived from haloperidol, protects against the damaging effects of ischemia/reperfusion (I/R) injury in vitro and in vivo. " 08/24/2012 - "Effects of N-n-butyl haloperidol iodide on the rat myocardial sarcoplasmic reticulum Ca(2+)-ATPase during ischemia/reperfusion." 05/15/2010 - "N-n-Butyl haloperidol iodide (F2), a novel compound derived from haloperidol, protects against the damaging effects of ischemia/reperfusion (I/R) injury in vitro and in vivo. " 06/01/2006 - "We have previously shown that N-n-butyl haloperidol iodide (F2) derived from haloperidol reduces ischemia/reperfusion-induced myocardial injury by blocking intracellular Ca2+ overload. "

Related Drugs and Biologics

1.	Calcium
2.	JNK Mitogen-Activated Protein Kinases
3.	Reactive Oxygen Species (Oxygen Radicals)
4.	Curcumin
5.	Haloperidol (Haldol)
6.	Ammonium Compounds
7.	Long Noncoding RNA
8.	Sarcoplasmic Reticulum Calcium-Transporting ATPases
9.	Catenins
10.	L-Type Calcium Channels (Dihydropyridine Receptor)