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F1F0-ATP synthase

in sunflowers; has been sequenced

Networked: 12 relevant articles (1 outcomes, 0 trials/studies)

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Bio-Agent Context: Research Results

Experts

1.	Huang, Wen-Yi: 2 articles (02/2014 - 01/2013)
2.	Jou, Mei-Jie: 2 articles (02/2014 - 01/2013)
3.	Peng, Tsung-I: 2 articles (02/2014 - 01/2013)
4.	Denny, William A: 1 article (01/2021)
5.	Armstrong, Jane: 1 article (01/2018)
6.	Campbell, Fiona: 1 article (01/2018)
7.	Costello, Eithne: 1 article (01/2018)
8.	Criddle, David N: 1 article (01/2018)
9.	Evans, Anthony: 1 article (01/2018)
10.	Haynes, Lee: 1 article (01/2018)

Related Diseases

1.	Osteosarcoma (Osteogenic Sarcoma) 02/01/2014 - "Here we found that a 1-h hypoxic preconditioning delayed apoptosis and improved cell survival after stimulation with various apoptotic inducers including H2O2, ionomycin, and arachidonic acid in mitochondrial DNA T8993G mutation (NARP) osteosarcoma 143B cybrids, an F1F0-ATP synthase defect cell model. "
2.	Neuronal Ceroid-Lipofuscinoses (Neuronal Ceroid Lipofuscinosis) 10/24/1997 - "Turnover of F1F0-ATP synthase subunit 9 and other proteolipids in normal and Batten disease fibroblasts." 07/01/1997 - "Polyunsaturated fatty acids reverse the lysosomal storage and accumulation of subunit 9 of mitochondrial F1F0-ATP synthase in cultured lymphoblasts from patients with Batten disease." 02/01/1996 - "Most forms of Batten Disease (BD), a group of neurodegenerative diseases, are characterized by the accumulation within lysosomes of the very hydrophobic protein subunit 9 of the mitochondrial F1F0-ATP synthase (F-ATPase). "
3.	Mitochondrial Diseases (Mitochondrial Disease) 01/12/2007 - "The molecular pathogenic mechanism of the human mitochondrial diseases neurogenic ataxia and retinitis pigmentosa and maternally inherited Leigh syndrome was determined in cultured human cells harboring homoplasmic T8993G/T8993C point mutations in the mitochondrial ATP6 gene, which encodes subunit 6 of the F1F0-ATP synthase. " 01/01/2013 - "mtDNA T8993G mutation-induced F1F0-ATP synthase defect augments mitochondrial dysfunction associated with hypoxia/reoxygenation: the protective role of melatonin."
4.	Leber Hereditary Optic Atrophy (Leber's Hereditary Optic Neuropathy) 07/28/1997 - "A lowered efficiency of oxidative phosphorylation was recently found in a Leber hereditary optic neuropathy (LHON) proband carrying a mutation in the mtDNA gene for subunit 6 of the membrane-bound F0 segment of the F1F0-ATP synthase [9]. "
5.	Neurodegenerative Diseases (Neurodegenerative Disease) 02/01/1996 - "Most forms of Batten Disease (BD), a group of neurodegenerative diseases, are characterized by the accumulation within lysosomes of the very hydrophobic protein subunit 9 of the mitochondrial F1F0-ATP synthase (F-ATPase). "

Related Drugs and Biologics

1.	Mitochondrial DNA (mtDNA)
2.	Arachidonic Acid (Vitamin F)
3.	Ionomycin
4.	Protein Subunits (Protein Subunit)
5.	Proteolipids
6.	Melatonin
7.	Fluorouracil (Carac)
8.	Unsaturated Fatty Acids (Polyunsaturated Fatty Acids)
9.	Enzymes
10.	Adenosine Triphosphate (ATP)