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magnolialide

a eudesmanolide found in Laurus nobilis (Lauraceae) and Cichorium intybus (Asteraceae)

Networked: 2 relevant articles (0 outcomes, 0 trials/studies)

Bio-Agent Context: Research Results

Organic Chemicals: 133
- Hydrocarbons: 1713
  - Terpenes: 1461
    - Sesquiterpenes: 1370
      - magnolialide: 2
- Lactones: 1549
  - magnolialide: 2

Experts

1.	Ho Kim, Kyeong: 1 article (09/2013)
2.	Lee, Sooryun: 1 article (09/2013)
3.	Lee, Taehun: 1 article (09/2013)
4.	Mar, Woongchon: 1 article (09/2013)
5.	Oh, Ki-Bong: 1 article (09/2013)
6.	Shin, Jongheon: 1 article (09/2013)
7.	Han, Y N: 1 article (08/2000)
8.	Kim, J I: 1 article (08/2000)
9.	Lee, K T: 1 article (08/2000)
10.	Miyamoto, K: 1 article (08/2000)

Related Diseases

1.	Leukemia 08/01/2000 - "Magnolialide inhibits the growth of several tumor cell lines and appears to induce differentiation of human leukemia HL-60 and U-937 cells to monocyte/macrophage-like cells. " 08/01/2000 - "Differentiation-inducing effect of magnolialide, a 1 beta-hydroxyeudesmanolide isolated from Cichorium intybus, on human leukemia cells."
2.	Immediate Hypersensitivity 09/16/2013 - "These results suggest that the magnolialide might be a candidate for the treatment of IgE-mediated hypersensitivity responses such as atopic dermatitis and asthma by inhibiting mast cell degranulation, the IL-4 production, and IL-5-dependent early B cell proliferation, key factors in the development and amplification of type I hypersensitivity reactions."
3.	Atopic Dermatitis (Atopic Eczema) 09/16/2013 - "These results suggest that the magnolialide might be a candidate for the treatment of IgE-mediated hypersensitivity responses such as atopic dermatitis and asthma by inhibiting mast cell degranulation, the IL-4 production, and IL-5-dependent early B cell proliferation, key factors in the development and amplification of type I hypersensitivity reactions."
4.	Asthma (Bronchial Asthma) 09/16/2013 - "These results suggest that the magnolialide might be a candidate for the treatment of IgE-mediated hypersensitivity responses such as atopic dermatitis and asthma by inhibiting mast cell degranulation, the IL-4 production, and IL-5-dependent early B cell proliferation, key factors in the development and amplification of type I hypersensitivity reactions."

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