CDR3.AME(82-89)

a synthetic CD4 exocyclic CDR3.AME(82-89); inhibits NF-kappaB nuclear translocation, HIV-1 promoter activation, and viral gene expression

Networked: 1 relevant articles (0 outcomes, 0 trials/studies)

1.	Infections 02/01/1997 - "The exocyclic CDR3.AME(82-89), derived from the CDR3 (residues 82-89) region of CD4 D1 domain, specifically inhibited binding of recombinant gp120 to both recombinant soluble CD4, and CD4+ Jurkat cells, and blocked syncytium formation and virus particle production caused by HIV-1 infection. "

1.	recombinant soluble CD4