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hexacyanoferrate III (ferricyanide)

RN given refers to parent cpd
Also Known As:
ferricyanide; ferric hexacyanide; iron-hexacyanide
Networked: 28 relevant articles (0 outcomes, 1 trials/studies)

Bio-Agent Context: Research Results

Experts

1. Gomez-Sarosi, Luis A: 1 article (01/2020)
2. Rieber, Manuel: 1 article (01/2020)
3. Rieber, Mary Strasberg: 1 article (01/2020)
4. Alves, Clayton Q: 1 article (01/2017)
5. Batista, Larissa F: 1 article (01/2017)
6. David, Jorge M: 1 article (01/2017)
7. Mafud, Ana C: 1 article (01/2017)
8. Mascarenhas, Yvonne P: 1 article (01/2017)
9. Medeiros, Jand V R: 1 article (01/2017)
10. Nicolau, Lucas A D: 1 article (01/2017)

Related Diseases

1. Neoplasms (Cancer)
05/21/2014 - "The chronoamperometric method with ferricyanide and succinate mediators is an efficient, alternative method for assessing the viability of primary hepatocytes which can be completed in 20 min. Succinate did not provide an efficient electron shuttle between cytosolic respiratory redox activity of cancer cells and extracellular ferricyanide, an effect that may be useful for distinguishing hepatocarcinoma cells from healthy hepatocytes. "
06/01/1995 - "It is suggested that NADH produced mainly by glycolysis is utilized as the substrate (electron donor) for transmembrane ferricyanide reduction, and the proton pumping activity in the cancer cells is coupled to the transmembrane NADH-ferricyanide redox enzyme system. "
06/01/1995 - "By using resonance energy transfer assay (RET) and fluorescence microscopy we show experimental evidence that membrane fusion of Ehrlich ascites of mastocarcinoma cells with liposomes could be induced by the proton translocation activity associated with NADH-ferricyanide redox enzyme of transplasma membrane of cancer cells. "
03/01/1971 - "In our studies, enzymes believed to be directly associated with the electron flow chain of NADPH, ferricyanide reduction, cytochrome c, cytochrome P-450 and substrate hydroxylation have been observed in livers obtained from normal, tumor-bearing and whole body irradiated rats as well as in Morris hepatoma 7777 and dimethyl-amino-biphenyl induced breast tumors.A significant difference appeared to exist in the activity of NADPH oxidase, NADP-ferricyanide reductase and benzopyrene hydroxylase when normal liver was compared with the liver obtained from a breast-tumor-bearing animal. "
2. Experimental Liver Neoplasms
3. Breast Neoplasms (Breast Cancer)
4. Hypoxia (Hypoxemia)
05/24/1991 - "Complete signal recovery after air or ferricyanide oxidation suggested the formation of hydroxylamine during hypoxia. "
11/01/1982 - "Metmyoglobin reduction, in anoxia following oxidation with ferricyanide (MRA) and aerobically following oxidation with low pO(2) (ARA), were also determined. "
04/01/2000 - "Chemical hypoxia-evoked cell death and LDH release were counteracted by the ferricyanide moiety of the SNP molecule, K3Fe(CN)6, and by ferric chloride (FeCl3), and this effect was counteracted by CB-DMB. "
02/15/2008 - "If the transmembrane potential under anoxia was supported by exogenous ATP or ferricyanide, the permeabilization of mitochondrial membranes by menadione or lucigenin was the same as under normoxia or even more pronounced. "
01/01/2020 - "-a) proliferating B16 melanoma with 10% serum-supplementation (10%S) normoxically express hypoxia inducible factor 1α (HIF1α) but lose tyrosinase, in contrast to those transiently exposed to (SF) serum-free medium, in which both HIF1α and tyrosinase are co-expressed; b) in contrast to the resistance to SNP toxicity in (SF) cells with higher tyrosinase expression, those in (10%S) are killed by iron from nitroprusside/ferricyanide (SNP) irrespective of exogenous H2O2, in a reaction antagonized by the anti-oxidant and MEK inhibitor UO126; c) Moreover, under transient serum depletion, SNP cooperates with hypoxia (1.5% oxygen), prolonging B16 melanoma (SF) survival; d) the hypoxia mimetic CoCl2 inhibits proliferation-associated cyclin A, irrespective of SNP, in (10%S) cells or in transiently serum-depleted (SF) cells. "
5. Poisoning

Related Drugs and Biologics

1. Cytochromes c (Cytochrome c)
2. Enzymes
3. ferricyanide reductase
4. NADPH Oxidases (NAD(P)H oxidase)
5. NADP (NADPH)
6. Cytochrome P-450 Enzyme System (Cytochrome P450)
7. Benzopyrene Hydroxylase
8. diphenyl (biphenyl)
9. Solutions
10. Protons (Proton)

Related Therapies and Procedures

1. Microelectrodes