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Neuropsychiatric Symptoms and Tick-Borne Diseases.

Abstract
In North America, Lyme disease (LD) is primarily caused by the spirochetal bacterium Borrelia burgdorferi, transmitted to humans by Ixodes species tick bites, at an estimated rate of 476,000 patients diagnosed per year. Acute LD often manifests with flu-like symptoms and an expanding rash known as erythema migrans (EM) and less often with neurologic, neuropsychiatric, arthritic, or cardiac features. Most acute cases of Lyme disease are effectively treated with antibiotics, but 10-20% of individuals may experience recurrent or persistent symptoms. This chapter focuses on the neuropsychiatric aspects of Lyme disease, as these are less widely recognized by physicians and often overlooked. Broader education about the potential complexity, severity, and diverse manifestations of tick-borne diseases is needed.
AuthorsShannon L Delaney, Lilly A Murray, Brian A Fallon
JournalCurrent topics in behavioral neurosciences (Curr Top Behav Neurosci) Vol. 61 Pg. 279-302 ( 2023) ISSN: 1866-3370 [Print] Germany
PMID36512289 (Publication Type: Journal Article)
Copyright© 2022. The Author(s), under exclusive license to Springer Nature Switzerland AG.
Topics
  • Animals
  • Humans
  • Lyme Disease (diagnosis, microbiology)
  • Tick-Borne Diseases (microbiology)
  • Erythema Chronicum Migrans
  • Ixodes (microbiology)

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