Regression of a large congenital hepatic arteriovenous malformation.

Abstract

Congenital hepatic arteriovenous malformations are rarely seen in association with persistent neonatal pulmonary hypertension. We report the case of a full-term female newborn who presented with heart failure and respiratory distress soon after birth. Echocardiographic investigation revealed severe persistent pulmonary hypertension and patent ductus arteriosus. Here we report spontaneous regression in size of both the feeder vessel and the vascular bed of the congenital hepatic arteriovenous malformation. We postulate that our conservative use of oral heart failure therapy, in the form of diuretic agents and captopril, decreased the congestion and diameter of the affected vessels.

• Authors: Hala Mounir Agha, Rania Zakaria, Fatma Alzahraa Mostafa, Hala Hamza
• Journal: Texas Heart Institute journal (Tex Heart Inst J) Vol. 42 Issue 2 Pg. 184-7 (Apr 2015) ISSN: 1526-6702 [Electronic] United States
• PMID: 25873838 (Publication Type: Case Reports, Journal Article)

Chemical References

• Diuretics
• Furosemide
• Captopril

Topics

• Arteriovenous Malformations (complications, diagnosis)
• Captopril (therapeutic use)
• Diuretics (therapeutic use)
• Female
• Furosemide (therapeutic use)
• Heart Failure (drug therapy, etiology)
• Hepatic Veins (pathology)
• Humans
• Hypertrophy
• Infant, Newborn
• Liver (blood supply)
• Persistent Fetal Circulation Syndrome (etiology)
• Remission, Spontaneous