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How far can mice carry autism research?

Abstract
In the face of growing controversy about the utility of genetic mouse models of human disease, Rothwell et al. report on a shared mechanism by which two different neuroligin-3 mutations, associated with autism spectrum disorders in humans, produce an enhancement in motor learning. The open question is how much we can learn about human ills from such models.
AuthorsSteven E Hyman
JournalCell (Cell) Vol. 158 Issue 1 Pg. 13-4 (Jul 03 2014) ISSN: 1097-4172 [Electronic] United States
PMID24995974 (Publication Type: Journal Article, Comment)
CopyrightCopyright © 2014 Elsevier Inc. All rights reserved.
Chemical References
  • Cell Adhesion Molecules, Neuronal
  • Membrane Proteins
  • Nerve Tissue Proteins
  • neuroligin 3
Topics
  • Animals
  • Autistic Disorder (genetics, physiopathology)
  • Cell Adhesion Molecules, Neuronal (genetics)
  • Humans
  • Membrane Proteins (genetics)
  • Nerve Tissue Proteins (genetics)

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