Abstract | BACKGROUND & AIMS: METHODS: RESULTS: CONCLUSIONS:
NFATc3 regulates trypsinogen activation, inflammation, and pancreatic tissue damage during development of AP in mice and might be a therapeutic target.
|
Authors | Darbaz Awla, Anna V Zetterqvist, Aree Abdulla, Cristina Camello, Lisa M Berglund, Peter Spégel, Maria J Pozo, Pedro J Camello, Sara Regnér, Maria F Gomez, Henrik Thorlacius |
Journal | Gastroenterology
(Gastroenterology)
Vol. 143
Issue 5
Pg. 1352-1360.e7
(Nov 2012)
ISSN: 1528-0012 [Electronic] United States |
PMID | 22841788
(Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
|
Copyright | Copyright © 2012 AGA Institute. Published by Elsevier Inc. All rights reserved. |
Chemical References |
- A 285222
- Chemokine CXCL2
- Cxcl2 protein, mouse
- NFATC Transcription Factors
- Nfatc3 protein, mouse
- Pyrazoles
- Taurocholic Acid
- Trypsinogen
- Peroxidase
- Amylases
|
Topics |
- Acinar Cells
(drug effects, metabolism)
- Amylases
(blood, drug effects)
- Animals
- Aorta
(metabolism)
- Cell Nucleus
(metabolism)
- Chemokine CXCL2
(drug effects, metabolism)
- Lung
(metabolism)
- Mice
- NFATC Transcription Factors
(antagonists & inhibitors, drug effects, genetics, metabolism)
- Neutrophils
(drug effects, physiology)
- Pancreatitis
(chemically induced, genetics, metabolism, pathology)
- Peroxidase
(drug effects, metabolism)
- Pyrazoles
(pharmacology)
- Signal Transduction
- Spleen
(metabolism)
- Statistics, Nonparametric
- Taurocholic Acid
- Trypsinogen
(drug effects, metabolism)
|