Abstract | UNLABELLED: CONCLUSION: This study revealed that LCA induced disruption of phospholipid/ sphingolipid homeostasis through TGF-β signaling and that serum LPC is a biomarker for biliary injury.
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Authors | Tsutomu Matsubara, Naoki Tanaka, Andrew D Patterson, Joo-Youn Cho, Kristopher W Krausz, Frank J Gonzalez |
Journal | Hepatology (Baltimore, Md.)
(Hepatology)
Vol. 53
Issue 4
Pg. 1282-93
(Apr 2011)
ISSN: 1527-3350 [Electronic] United States |
PMID | 21480330
(Publication Type: Journal Article, Research Support, N.I.H., Intramural, Research Support, Non-U.S. Gov't)
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Copyright | American Association for the Study of Liver Diseases. |
Chemical References |
- Ceramides
- Lysophosphatidylcholines
- Phospholipids
- RNA, Messenger
- Sphingolipids
- Sphingomyelins
- Transforming Growth Factor beta
- Lithocholic Acid
- 1-Acylglycerophosphocholine O-Acyltransferase
- Smpd3 protein, mouse
- Sphingomyelin Phosphodiesterase
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Topics |
- 1-Acylglycerophosphocholine O-Acyltransferase
(biosynthesis)
- Animals
- Ceramides
(biosynthesis)
- Cholestasis
- Female
- Homeostasis
(drug effects)
- Lithocholic Acid
(blood, toxicity)
- Lysophosphatidylcholines
(blood)
- Metabolome
- Mice
- Mice, Inbred C57BL
- Phospholipids
(metabolism)
- RNA, Messenger
(metabolism)
- Sphingolipids
(metabolism)
- Sphingomyelin Phosphodiesterase
(biosynthesis)
- Sphingomyelins
(blood)
- Transforming Growth Factor beta
(physiology)
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