Abstract |
Cerebral arteriovenous malformations (AVMs) have abnormally enlarged arteries and veins prone to spontaneous hemorrhage. Immediately following surgical excision of a cerebral AVM, even normal brain tissue surrounding the lesion is subject to hemorrhage, a phenomenon termed normal perfusion pressure breakthrough ( NPPB) syndrome. According to this theory, arteries supplying cerebral AVMs become dilated and lose their capacity to dilate or constrict to autoregulate pressure. Acutely after removal of a cerebral AVM, excessive blood pressure in these arterial feeders can cause normal brain tissue to bleed. However, this theory remains controversial. We present a patient with a cerebral AVM that demonstrated cerebrovascular reactivity and argues against an assumption underlying the theory of NPPB syndrome.
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Authors | Matthew D Alexander, E Sander Connolly, Philip M Meyers |
Journal | World journal of radiology
(World J Radiol)
Vol. 2
Issue 6
Pg. 230-2
(Jun 28 2010)
ISSN: 1949-8470 [Electronic] United States |
PMID | 21160635
(Publication Type: Journal Article)
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