Abstract | BACKGROUND: AIM: METHODS: RESULTS: CONCLUSION: Although the expression of ASBT was similarly diminished in both gallstone and Crohn's disease, subclinical ileal inflammation does not appear to be relevant in gallstone patients. The mechanisms of transcriptional repression of ASBT in both diseases are apparently different.
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Authors | Arthur Holzer, Simone Harsch, Olga Renner, André Strohmeyer, Silke Schimmel, Jan Wehkamp, Peter Fritz, Eduard F Stange |
Journal | Digestion
(Digestion)
Vol. 78
Issue 1
Pg. 52-9
( 2008)
ISSN: 1421-9867 [Electronic] Switzerland |
PMID | 18832832
(Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
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Copyright | 2008 S. Karger AG, Basel. |
Chemical References |
- Inflammation Mediators
- Interleukin-1beta
- Interleukin-8
- Organic Anion Transporters, Sodium-Dependent
- Proto-Oncogene Proteins c-fos
- Proto-Oncogene Proteins c-jun
- RNA, Messenger
- Symporters
- Tumor Necrosis Factor-alpha
- sodium-bile acid cotransporter
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Topics |
- Adult
- Aged
- Case-Control Studies
- Crohn Disease
(metabolism, pathology)
- Female
- Gallstones
(metabolism)
- Humans
- Ileitis
(metabolism, pathology)
- Ileum
(metabolism, pathology)
- Inflammation Mediators
(metabolism)
- Interleukin-1beta
(metabolism)
- Interleukin-8
(metabolism)
- Middle Aged
- Organic Anion Transporters, Sodium-Dependent
(metabolism)
- Proto-Oncogene Proteins c-fos
(metabolism)
- Proto-Oncogene Proteins c-jun
(metabolism)
- RNA, Messenger
(metabolism)
- Symporters
(metabolism)
- Tumor Necrosis Factor-alpha
(metabolism)
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