Abstract |
Our studies suggest that the fluctuation of HDC activity in fetal liver in late gestation is regulated by the plasma glucocorticoid level through the pituitary-adrenal system. Taken together, these results support the conclusion that glucocorticoid promotes a rapid increase in HDC synthesis in fetal liver histamine-forming cells, as well as in mouse mastocytoma P-815 cells and rat glandular stomachs.
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Authors | E Ohmori, N Imanishi, M Ohgoh, T Fukui, A Ichikawa |
Journal | Biochemical pharmacology
(Biochem Pharmacol)
Vol. 41
Issue 5
Pg. 844-7
(Mar 01 1991)
ISSN: 0006-2952 [Print] England |
PMID | 1847820
(Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
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Chemical References |
- Histamine
- Adrenocorticotropic Hormone
- Histidine Decarboxylase
- Corticosterone
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Topics |
- Adrenocorticotropic Hormone
(blood, metabolism)
- Animals
- Corticosterone
(blood, metabolism)
- Fetus
(enzymology)
- Histamine
(metabolism)
- Histidine Decarboxylase
(biosynthesis, blood)
- Liver
(embryology, enzymology)
- Pituitary-Adrenal System
(physiology)
- Rats
- Rats, Inbred Strains
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