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Klüver-Bucy syndrome following status epilepticus associated with hepatic encephalopathy.

Abstract
Described here is the case of a patient with liver cirrhosis who developed bilateral temporo-occipital lobe lesions on MRI and Klüver-Bucy syndrome following status epilepticus. Herpes encephalitis, paraneoplastic syndrome, Hashimoto's encephalopathy, reversible posterior leukoencephalopathy syndrome, mitochondrial encephalomyopathy, lactic acidosis, and strokelike episode syndrome were judged not to be involved on the basis of laboratory results. The possible cause of the temporo-occipital lesions on MRI in this patient was cortical damage related mainly to status epilepticus and partially to coexisting hepatic encephalopathy.
AuthorsKosuke Naito, Takao Hashimoto, Shu-ichi Ikeda
JournalEpilepsy & behavior : E&B (Epilepsy Behav) Vol. 12 Issue 2 Pg. 337-9 (Feb 2008) ISSN: 1525-5050 [Print] United States
PMID17980671 (Publication Type: Case Reports, Journal Article)
Chemical References
  • Adrenal Cortex Hormones
Topics
  • Adrenal Cortex Hormones (therapeutic use)
  • Electroencephalography
  • Female
  • Functional Laterality
  • Hepatic Encephalopathy (complications, drug therapy, pathology)
  • Humans
  • Kluver-Bucy Syndrome (drug therapy, etiology, pathology, physiopathology)
  • Liver Cirrhosis, Alcoholic (complications)
  • Magnetic Resonance Imaging
  • Middle Aged
  • Occipital Lobe (pathology, physiopathology)
  • Status Epilepticus (complications, pathology, physiopathology)
  • Temporal Lobe (pathology, physiopathology)
  • Treatment Outcome

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