| Abstract | AIM: To investigate if weight gain during adulthood has effects on the risk of developing impaired glucose tolerance (IGT) or Type 2 diabetes beyond effect of attained weight. RESEARCH DESIGN AND METHODS: Data were obtained from a longitudinal study of two cohorts: one of juvenile-onset obese (n = 248) and one of randomly selected control (n = 320) men, weighed at average ages of 20, 33, 44 and 51 years, respectively. RESULTS: For any given BMI, the risk of IGT was higher the greater the weight gain since age 20 (odds ratio of 1.10 per unit kg/m2 of BMI gain, confidence interval 1.03-1.17, P = 0.004), and weight gain during both the early and later ages contributed to the increased risk. Obese men, maintaining weight since age 20, had lower risk of IGT than non-obese men who became similarly obese by age 51. The risk of Type 2 diabetes increased by weight gain in early adult life, but not by more recent weight gain in the later periods, probably because of the development of Type 2 diabetes leading to weight loss. CONCLUSIONS: Independent of attained level of body weight in middle-aged men, weight gain is associated with increased risk of IGT, and is greater in those not overweight in childhood. |
| Authors | E Black, C Holst, A Astrup, S Toubro, S Echwald, O Pedersen, T I A Sørensen
(Affiliation: Department of Human Nutrition, Centre of Advanced Food Research, The Royal Veterinary and Agricultural University, Frederiksberg, Denmark.)
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| Journal | Diabetic medicine : a journal of the British Diabetic Association
(Diabet Med)
Vol. 22
Issue 9
Pg. 1199-205
(Sep 2005)
ISSN: 0742-3071 England |
| PMID | 16108849
(Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
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| Chemical References |
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| Topics |
- Adult
- Aging
(physiology)
- Blood Glucose
(analysis)
- Body Mass Index
- Diabetes Mellitus, Type 2
(epidemiology, etiology, physiopathology)
- Glucose Intolerance
(epidemiology, etiology, physiopathology)
- Glucose Tolerance Test
(methods)
- Humans
- Longitudinal Studies
- Male
- Middle Aged
- Obesity
(physiopathology)
- Prevalence
- Risk Factors
- Weight Gain
(physiology)
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