Miliaria crystallina in an intensive care setting.

Abstract

Drop-like, transient blisters of miliaria crystallina may develop with focal intensity of heat within the skin, such as occurs in tropical climates or during febrile episodes. Miliaria crystallina develops due to a transient poral closure of the sweat duct opening, resulting in obstruction of free flow of eccrine sweat and retention in a vesicle below the skin surface. Dual cholinergic and adrenergic sweat gland innervation is influenced by a variety of medications used in intensive care patients. We present two febrile intensive care patients in whom enhanced alpha-adrenergic stimulation of sweat gland myoepithelia may have led to miliaria crystallina.

• Authors: N Haas, F Martens, B M Henz
• Journal: Clinical and experimental dermatology (Clin Exp Dermatol) Vol. 29 Issue 1 Pg. 32-4 (Jan 2004) ISSN: 0307-6938 [Print] England
• PMID: 14723716 (Publication Type: Case Reports, Journal Article)

Chemical References

• Adrenergic alpha-Agonists
• Adrenergic beta-Agonists
• Clonidine
• Albuterol

Topics

• Adrenergic alpha-Agonists (adverse effects)
• Adrenergic beta-Agonists (adverse effects)
• Adult
• Albuterol (adverse effects)
• Clonidine (adverse effects)
• Critical Care (methods)
• Drug Eruptions (etiology)
• Female
• Humans
• Male
• Miliaria (chemically induced, therapy)