Abstract | BACKGROUND: Acute barium poisoning is most often the result of accidental or suicidal ingestion of the rodenticide, barium carbonate. We describe a trauma patient whose condition was complicated by severe acute barium toxicity from an explosion of the propellant, barium styphnate. In addition to critical injuries, the patient manifested classic signs of barium toxicity including repeated profound hypokalemia, cardiac arrhythmias, respiratory failure, prolonged gastrointestinal dysfunction, paralysis, myoclonus, hypertension, and profound lactic acidosis. METHODS: RESULTS: CONCLUSIONS: Considering the paucity of information concerning management of this life-threatening problem, the pathophysiology of barium toxicity, including the transfusion related hypokalemia, and its management is reviewed.
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Authors | Ira A Jacobs, Joseph Taddeo, Kathleen Kelly, Carl Valenziano |
Journal | American journal of industrial medicine
(Am J Ind Med)
Vol. 41
Issue 4
Pg. 285-8
(Apr 2002)
ISSN: 0271-3586 [Print] United States |
PMID | 11920972
(Publication Type: Case Reports, Journal Article)
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Copyright | Copyright 2002 Wiley-Liss, Inc. |
Chemical References |
- Barium Compounds
- barium styphnate
- Barium
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Topics |
- Accidents, Occupational
- Arrhythmias, Cardiac
(chemically induced)
- Barium
(poisoning)
- Barium Compounds
(administration & dosage)
- Explosions
- Humans
- Hypokalemia
(chemically induced)
- Male
- Middle Aged
- Poisoning
(therapy)
- Transfusion Reaction
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