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Persistence of ketosis despite suppression of lipolysis by 16,16-dimethylprostaglandin E2 in experimental diabetic ketoacidosis in rats.

AuthorsL Axelrod, S D Slowman, P Lloyd-Jones, D B Martin
JournalDiabetes (Diabetes) Vol. 28 Issue 10 Pg. 905-13 (Oct 1979) ISSN: 0012-1797 [Print] United States
PMID113273 (Publication Type: Journal Article, Research Support, U.S. Gov't, P.H.S.)
Chemical References
  • Acetoacetates
  • Fatty Acids, Nonesterified
  • Hydroxybutyrates
  • Insulin
  • Ketone Bodies
  • Prostaglandins E, Synthetic
  • 16,16-Dimethylprostaglandin E2
  • Glycerol
Topics
  • 16,16-Dimethylprostaglandin E2 (pharmacology)
  • Acetoacetates (blood)
  • Animals
  • Diabetes Mellitus, Experimental (metabolism)
  • Diabetic Ketoacidosis (metabolism)
  • Fatty Acids, Nonesterified (blood)
  • Glycerol (blood)
  • Hydroxybutyrates (blood)
  • Insulin (pharmacology)
  • Ketone Bodies (blood)
  • Kinetics
  • Lipid Mobilization (drug effects)
  • Male
  • Prostaglandins E, Synthetic (pharmacology)
  • Rats

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