Abstract | OBJECTIVE: DEVELOPMENT: CONCLUSION: The knowledge of the role of the ion channels in the epilepsies is allowing the design of new and more specific therapeutic strategies.
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Authors | J A Armijo, I de las Cuevas, J Adín |
Journal | Revista de neurologia
(Rev Neurol)
Vol. 30 Suppl 1
Pg. S25-41
(Jun 2000)
ISSN: 0210-0010 [Print] Spain |
Vernacular Title | Canales iónicos y epilepsia. |
PMID | 10904966
(Publication Type: English Abstract, Journal Article, Review)
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Chemical References |
- Anticonvulsants
- Calcium Channels
- Chloride Channels
- Ion Channels
- Ligands
- Potassium Channels
- Receptors, AMPA
- Receptors, GABA
- Receptors, N-Methyl-D-Aspartate
- Receptors, Nicotinic
- Sodium Channels
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Topics |
- Angelman Syndrome
(genetics, metabolism)
- Animals
- Anticonvulsants
(pharmacokinetics)
- Calcium Channels
(genetics, metabolism)
- Chloride Channels
(genetics, metabolism)
- Epilepsy
(genetics, metabolism)
- Gene Expression
(genetics)
- Humans
- Ion Channels
(genetics, metabolism)
- Kindling, Neurologic
(genetics, metabolism)
- Ligands
- Long QT Syndrome
(diagnosis, genetics, metabolism)
- Mice
- Point Mutation
(genetics)
- Potassium Channels
(genetics, metabolism)
- Receptors, AMPA
(genetics, metabolism)
- Receptors, GABA
(genetics, metabolism)
- Receptors, N-Methyl-D-Aspartate
(genetics, metabolism)
- Receptors, Nicotinic
(genetics, metabolism)
- Sodium Channels
(genetics, metabolism)
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