|1.||Viapiano, Mariano S: 7 articles (10/2014 - 08/2003)|
|2.||Matthews, Russell T: 5 articles (10/2014 - 08/2003)|
|3.||Hockfield, S: 5 articles (10/2001 - 01/2000)|
|4.||Tang, Xiufeng: 3 articles (10/2008 - 02/2003)|
|5.||Davies, Jeannette E: 3 articles (10/2008 - 02/2003)|
|6.||Hockfield, Susan: 3 articles (07/2008 - 08/2003)|
|7.||Gary, S C: 3 articles (10/2000 - 01/2000)|
|8.||Bi, Wenya Linda: 2 articles (10/2014 - 08/2005)|
|9.||Klekner, Almos: 2 articles (04/2012 - 01/2010)|
|10.||Hutóczki, Gábor: 2 articles (04/2012 - 01/2010)|
01/01/2012 - "Moreover, the role of brevican in the growth and progression of glioma was demonstrated by in vivo studies. "
01/01/2012 - "In this study, immunohistochemistry staining for brevican expression was performed in malignant gliomas and benign controls. "
01/01/2012 - "The aim of this study was to examine whether brevican is a predictor of glioma and its roles in glioma cell motility. "
09/01/2005 - "In the present study, we examined brevican-degrading activities of ADAMTS-1, -4 and -5 at the cellular level, and their expression and localization in human glioma tissues. "
01/01/2012 - "Our results provide evidence for the molecular and cellular mechanisms that may underlie the motility-promoting role of brevican in the progression of glioma. "
10/01/2001 - "These data suggest that up-regulation and proteolytic cleavage of BEHAB/brevican increase significantly the aggressiveness of glial tumors. "
10/01/2001 - "Histological examination showed that the BEHAB/brevican-transfected tumors were just as, if not more, aggressive than control tumors, even though the BEHAB/brevican tumors had been growing for only approximately two-thirds the time as long as control tumors. "
04/01/2001 - "Here, the authors present a review of the literature on glial tumor invasion by modulation of the ECM and propose a two-step model for BEHAB/brevican's role in this process."
04/01/2001 - "Glial tumor invasion: a role for the upregulation and cleavage of BEHAB/brevican."
10/01/1998 - "Cleavage of the BEHAB/brevican protein may increase invasion of tumor cells."
|3.||Brain Injuries (Brain Injury)
07/01/2000 - "In order to study a potential role of brevican in the glial reaction after brain injury, its expression was analysed following entorhinal cortex lesion in rats (12 h, 1, 2, 4, 10, 14 and 28 days and 6 months post lesion). "
05/01/2005 - "The effect of hypoxic-ischemic brain injury in perinatal rats on the abundance and proteolysis of brevican and NG2."
04/01/2001 - "BEHAB/brevican expression is also upregulated during periods of increased glial cell motility in development and following brain injury. "
09/01/2013 - "Chondroitin sulfate proteoglycans (CSPGs)-neuorcan, brevican, versican, aggrecan and phosphacan-are unregulated in the extracellular matrix after brain injury, and their degradation enhances plasticity of the brain. "
|4.||Melanoma (Melanoma, Malignant)
|5.||Spinal Cord Injuries (Spinal Cord Injury)
01/01/2014 - "ADAMTS4 and ADAMTS5 knockout mice are protected from versican but not aggrecan or brevican proteolysis during spinal cord injury."
08/01/2003 - "After spinal cord injury, neurocan, brevican, and versican immunolabeling increased within days in injured spinal cord parenchyma surrounding the lesion site and peaked at 2 weeks. "
10/01/2008 - "Decorin suppresses neurocan, brevican, phosphacan and NG2 expression and promotes axon growth across adult rat spinal cord injuries. "
03/01/2004 - "Decorin suppresses neurocan, brevican, phosphacan and NG2 expression and promotes axon growth across adult rat spinal cord injuries."
10/15/2003 - "Immunohistochemical analysis showed dense labeling of the CSPGs NG2, brevican, neurocan, versican, and phosphacan at the host-lesion interface after spinal cord injury (SCI). "
|3.||Messenger RNA (mRNA)
|4.||Complementary DNA (cDNA)
|6.||Class 5 Receptor-Like Protein Tyrosine Phosphatases
|8.||Hyaluronic Acid (Hyaluronan)
|9.||Chondroitin Sulfate Proteoglycans
|3.||Infusion Pumps (Infusion Pump)