|1.||Bruns, Christiane J: 2 articles (02/2015 - 03/2014)|
|2.||Camaj, Peter: 2 articles (02/2015 - 03/2014)|
|3.||Nelson, Peter J: 2 articles (02/2015 - 03/2014)|
|4.||Jäckel, Carsten: 2 articles (02/2015 - 03/2014)|
|5.||Jauch, Karl-Walter: 2 articles (02/2015 - 03/2014)|
|6.||Thoms, Brendan L: 2 articles (11/2013 - 07/2010)|
|7.||Pinho, Andreia V: 1 article (11/2015)|
|8.||Mawson, Amanda: 1 article (11/2015)|
|9.||Grimont, Adrien: 1 article (11/2015)|
|10.||Rooman, Ilse: 1 article (11/2015)|
04/01/2009 - "Recent evidence indicates that the antiproliferative effects of RA are mediated by the transcription factor SOX9 in human cancer cell lines. "
04/01/2004 - "Two cases of ovarian Sertoli-stromal cell tumor were examined for expression of a transcription factor, SOX9, which plays an essential role in Sertoli cell differentiation, downstream of Sry, as in the testis. "
04/01/2004 - "Expression of a transcription factor, SOX9, in Sertoli-stromal cell tumors of the ovary."
10/01/2010 - "The transcription factor Sox9 is known to play a crucial role in normal chondrogenesis, and antibodies against Sox9 have been proposed as a diagnostic tool for neoplasms with chondroid differentiation. "
06/01/2014 - "Being overexpressed and regulated by NF-κB in different types of cancers, the transcription factor SOX9 was also decreased at the nuclear level by fucoxanthin and fucoxanthinol in MDA-MB-231. "
05/01/2003 - "Mutations in the transcription factor SOX9 give rise to campomelic dysplasia, a syndrome characterized by skeletal abnormalities and XY sex reversal. "
07/27/2001 - "Human mutations in the transcription factor SOX9 cause campomelic dysplasia/autosomal sex reversal. "
12/01/2013 - "Heterozygous loss-of-function coding-sequence mutations of the transcription factor SOX9 cause campomelic dysplasia, a rare skeletal dysplasia with congenital bowing of long bones (campomelia), hypoplastic scapulae, a missing pair of ribs, pelvic, and vertebral malformations, clubbed feet, Pierre Robin sequence (PRS), facial dysmorphia, and disorders of sex development. "
06/15/2003 - "Loss of DNA-dependent dimerization of the transcription factor SOX9 as a cause for campomelic dysplasia."
02/20/2015 - "The transcription factor SOX9, which is linked to regulation of hypoxia-related genes, was identified as a key mediator of upregulation of the oncogene IFIT3 and thereby sustaining a "pseudoinflammatory" cellular condition. "
03/01/2014 - "A subset of these transcripts were regulated by the transcription factor SOX9 in the aggressive-metastatic cells, but driven by hypoxia-inducible factor-1α (HIF-1α) in the parental nonmetastatic cell line. "
07/02/2010 - "We have previously shown that human chondrocytes enhance cartilage matrix synthesis in response to hypoxia specifically through hypoxia-inducible factor 2alpha (HIF-2alpha)-mediated up-regulation of master regulator transcription factor SOX9, which in turn drives expression of the main cartilage-specific extracellular matrix genes. "
11/01/2013 - "Using recombinant PTHrP, siRNA-mediated depletion of endogenous PTHrP and by blocking signalling through its receptor [PTHR1 (PTHrP receptor 1)], we show that hypoxia-induced PTHrP is a positive regulator of the key cartilage transcription factor SOX9 [SRY (sex determining region on the Y chromosome)-box 9], leading to increased COL2A1 (collagen type II, α1) expression. "
09/01/2014 - "COX-2 was expressed in DCAMKL1-positive tuft cells in Cdx2- and DCAMKL1-transgenic mouse stomachs, whereas the Sox9 transcription factor was ubiquitously expressed in gastric carcinomas, including COX-2-positive cells. "
03/15/2005 - "In this report, we show that the transcription factor SOX9 down-regulates CEA gene expression and, as a probable consequence, induces apoptosis in the human colon carcinoma cell line HT29Cl.16E."
|5.||Intervertebral Disk Degeneration
|2.||Type 1 Parathyroid Hormone Receptor (Parathyroid Hormone Receptor)
|3.||Cysteine Proteases (Cysteine Protease)
|6.||Parathyroid Hormone-Related Protein
|7.||Small Interfering RNA (siRNA)
|10.||Fibroblast Growth Factors (Fibroblast Growth Factor)