|1.||Delivoria-Papadopoulos, Maria: 10 articles (08/2011 - 04/2002)|
|2.||Ashraf, Qazi M: 8 articles (08/2011 - 04/2002)|
|3.||Mishra, Om P: 7 articles (08/2008 - 04/2002)|
|4.||Zubrow, Alan B: 4 articles (12/2006 - 12/2002)|
|5.||Fritz, Karen I: 4 articles (01/2006 - 12/2002)|
|6.||Mishra, Om Prakash: 3 articles (08/2011 - 12/2006)|
|7.||Zhuo, Min: 3 articles (10/2008 - 01/2004)|
|8.||Koga, Tomohiro: 2 articles (12/2015 - 10/2012)|
|9.||Mizui, Masayuki: 2 articles (12/2015 - 10/2012)|
|10.||Tsokos, George C: 2 articles (12/2015 - 10/2012)|
01/01/2014 - "In conclusion, our preliminary data show that concurrent administration of Src kinase inhibitor in combination with induction of whole body hypothermia results in augmented neuroprotection as indicated by further attenuation of hypoxic-ischemic induced CaM kinase IV activation and improvement in neuronal membrane integrity compared to hypothermia alone. "
01/01/2014 - "To this aim we assessed the levels of CaM Kinase IV activity as well as the levels of Na+-K+-ATPase in the Cerebral Cortex of Newborn Piglets exposed experimental hypoxia that were treated with hypothermia with or without concomitant PP2 administration. "
03/17/2011 - "Brain tissue energy dependence of CaM kinase IV cascade activation during hypoxia in the cerebral cortex of newborn piglets."
08/08/2008 - "Mechanism of Ca2+-influx and Ca2+/calmodulin-dependent protein kinase IV activity during in utero hypoxia in cerebral cortical neuronal nuclei of the guinea pig fetus at term."
12/01/2006 - "However, hypoxia-induced increase in CaM Kinase IV activity decreased during the post-Hx reox. "
09/30/2005 - "Previously we have demonstrated that in utero hypoxia results in increased nuclear Ca(2+)-influx and increased CaM kinase IV activity in neuronal nuclei of the guinea pig fetus. "
01/01/2004 - "There was a significant difference among CaM kinase IV activity (pmol/mg protein/min) in normoxia (1270.80+/-126.1), hypoxia (2680.80+/-136.7; P<0.05 vs normoxia), NNLA-pretreated hypoxia (1666.00+/-154.8; P<0.05 vs normoxia, P<0.05 vs hypoxia), and 7-NINA-pretreated hypoxic (1712.9+/-231.5; P=NS vs normoxia, P<0.05 vs hypoxia). "
|3.||Memory Disorders (Memory Loss)
10/01/2008 - "Upregulation of calcium/calmodulin-dependent protein kinase IV improves memory formation and rescues memory loss with aging."
10/01/2008 - "Previous studies have suggested that calcium/calmodulin-dependent protein kinase IV (CaMKIV) functions as a positive regulator for memory formation and that age-related memory deficits are the result of dysfunctional signaling pathways mediated by cAMP response element-binding protein (CREB), the downstream transcription factor of CaMKIV. "
|4.||Hepatocellular Carcinoma (Hepatoma)
|5.||Cardiomegaly (Heart Hypertrophy)
|2.||Cyclic AMP Response Element-Binding Protein (Cyclic AMP-Responsive DNA-Binding Protein)
|3.||Calmodulin (Calcium-Dependent Activator Protein)
|5.||Magnesium Sulfate (Sulfate, Magnesium)
|6.||Transcription Factors (Transcription Factor)
|7.||Calcium-Calmodulin-Dependent Protein Kinases
|9.||Adenosine Triphosphate (ATP)
|10.||Nitric Oxide Synthase (NO Synthase)