|1.||Li, Mingjiang: 2 articles (03/2015 - 08/2013)|
|2.||Yan, Lei: 2 articles (03/2015 - 08/2013)|
|3.||Zhao, Xingbo: 2 articles (03/2015 - 08/2013)|
|4.||Wang, Chong: 2 articles (03/2015 - 08/2013)|
|5.||Zhang, Hui: 2 articles (03/2015 - 08/2013)|
|6.||Guo, Qiufen: 2 articles (03/2015 - 08/2013)|
|7.||Kim, Se Hyun: 2 articles (12/2012 - 04/2012)|
|8.||Kim, Yong Sik: 2 articles (12/2012 - 04/2012)|
|9.||Ahn, Yong Min: 2 articles (12/2012 - 04/2012)|
|10.||Sato, T: 1 article (11/2015)|
11/01/2015 - "In this study, we found that BAALC induced cell-cycle progression of leukemia cells by sustaining extracellular signal-regulated kinase (ERK) activity through an interaction with a scaffold protein MEK kinase-1 (MEKK1), which inhibits the interaction between ERK and MAP kinase phosphatase 3 (MKP3/DUSP6). "
11/01/2013 - "Expression of CSF1 (colony stimulating factor for macrophages), carbonic anhydrase 9 (CA9), epithelial growth factor receptor (EGFR), dual specificity phosphatase 6 (DUSP6), v-erb-b2 erythroblastic leukemia viral oncogene homolog 3 (ERBB3), monocyte to macrophage differentiation-associated (MMD), lymphocyte-specific protein tyrosine kinase (LCK) and signal transducer and activator of transcription 1 (STAT1) was assessed in SCC, AC, and in normal lung by quantitative reverse transcriptase - polymerase chain reaction (qRT-PCR). "
12/01/2014 - "Dual specificity phosphatase 6 (DUSP6) is expressed at low levels in numerous types of human cancer. "
01/01/2012 - "Tumor suppressor dual-specificity phosphatase 6 (DUSP6) impairs cell invasion and epithelial-mesenchymal transition (EMT)-associated phenotype."
08/20/2013 - "Dual specificity phosphatase 6 (DUSP6) is a member of a family of mitogen-activated protein kinase phosphatases that dephosphorylates and inhibits activated ERK1/2. Dual specificity phosphatase 6 is dynamically regulated in developmental and pathological conditions such as cancer. "
08/01/2015 - "Whole exome sequencing and additional deep-sequencing analyses revealed the enhanced accumulation of somatic mutations in various genes, including dual specificity phosphatase 6 (Dusp6), early growth response 1 (Egr1) and inhibitor of DNA binding 2 (Id2), which are putative tumor suppressors, in AID-expressing liver with TAA-mediated hepatic inflammation. "
03/01/2015 - "We previously found that Dual-specificity phosphatase 6 (Dusp6) over-expression enhanced the growth-promoting effect of estrogen in endometrial adenocarcinoma cells. "
08/25/2013 - "Dual-specificity phosphatase 6 (Dusp6), a negative regulator of FGF2/ERK1/2 signaling, enhances 17β-estradiol-induced cell growth in endometrial adenocarcinoma cell."
|4.||Thymoma (Thymic Carcinoma)
07/01/2005 - "Upregulated genes in cell lines with mutations included dual-specificity phosphatase 6 (DUSP6), sprouty 2 (SPRY2), v-akt murine thymoma viral oncogene homolog 3 (AKT3) and matrix metalloproteinase 14 (MMP14); downregulated genes included interleukin 18 (IL18), Krüppel-like factor 5 (KLF5) and inhibitor of DNA binding 2 (ID2). "
|2.||DNA (Deoxyribonucleic Acid)
|3.||Mitogen-Activated Protein Kinase Phosphatases
|4.||Matrix Metalloproteinase 14 (MT1-MMP)
|5.||STAT1 Transcription Factor
|6.||Interleukin-18 (Interleukin 18)
|7.||Fibroblast Growth Factor 2 (Basic Fibroblast Growth Factor)
|9.||RNA-Directed DNA Polymerase (Reverse Transcriptase)
|10.||Protein-Tyrosine Kinases (Tyrosine Kinase)