|1.||Abe, Masahiro: 4 articles (04/2008 - 09/2002)|
|2.||Ishizu, Takaaki: 3 articles (12/2008 - 01/2005)|
|3.||Kira, Jun-ichi: 3 articles (12/2008 - 01/2005)|
|4.||Matsumoto, Toshio: 3 articles (04/2008 - 09/2002)|
|5.||Van Damme, Jo: 3 articles (09/2006 - 10/2002)|
|6.||Pawlak, Krystyna: 3 articles (12/2004 - 01/2004)|
|7.||Pawlak, Dariusz: 3 articles (12/2004 - 01/2004)|
|8.||Lusso, Paolo: 2 articles (01/2010 - 06/2008)|
|9.||Velikokhatnaya, Lyudmila: 2 articles (03/2009 - 04/2007)|
|10.||Lokshin, Anna E: 2 articles (03/2009 - 04/2007)|
01/01/2010 - "Nef-specific CD45RA+ CD8+ T cells secreting MIP-1beta but not IFN-gamma are associated with nonprogressive HIV-1 infection."
04/01/2001 - "We conclude that neutrophils fail to prevent viral infection by IFN production but probably possess alternative mechanisms, such as secreting MIP-1beta in response to viruses."
04/01/2001 - "maxima infection but at a lower magnitude except for those encoding MIP-1beta and MGF. "
12/05/2000 - "Higher macrophage inflammatory protein (MIP)-1alpha and MIP-1beta levels from CD8+ T cells are associated with asymptomatic HIV-1 infection."
08/01/1998 - "The CD8(+) IEL were the primary producers of IFN-gamma and MIP-1beta and were found to retain their potential to produce both IFN-gamma and MIP-1beta through the entire course of SIV infection. "
07/01/2009 - "The concentration of OPN was significantly elevated in RA patients and appeared to correlate with the serum levels of inflammation markers and increased expression of MCP-1 or MIP-1beta in monocytes in RA patients. "
10/01/2006 - "Our data suggest that CCL4/MIP-1beta plays a role in preventing excessive inflammation and pathology rather than in controlling parasite replication."
10/01/2006 - "The expression of CCL2/MCP-1 and CCL4/MIP-1beta correlated closely with the kinetics of myocardial inflammation. "
11/01/2003 - "These observations suggest that IL-1beta-mediated up-regulation of MIP-1beta production in the hepatic cells may contribute a critical mechanism for continuous recruitment of inflammatory cell to liver and maintenance of inflammation."
02/15/2003 - "Most importantly, we demonstrated that SP and NK-1R presented in NT2-N cells are functionally involved in the regulation of macrophage inflammatory protein 1 beta (MIP-1beta), an important beta-chemokine participating in the activation and directional migration of immune cells to sites of central nervous systems (CNS) inflammation. "
07/01/2009 - "Furthermore, we examined the efficacy of MIP-1beta as a predictor of stroke and cardiovascular events in hypertensive patients. "
07/01/2009 - "In our clinical study, patients with the highest quartile of MIP-1beta showed a higher risk of stroke and cardiovascular events by a Cox proportional-hazards model. "
07/01/2009 - "We designed an in vitro investigation to examine the role of MIP-1beta on the development of atherosclerosis, including cell adhesion involving CAMs and ROS production, compared with angiotensin II. Furthermore, we investigated the prognostic impact of MIP-1beta on stroke and cardiovascular events in hypertensive patients in a small cohort study."
02/01/1998 - "We evaluated mRNA expression of the CXC chemokine interleukin (IL)-8, and the CC chemokines monocyte chemoattractant protein-1, macrophage inflammatory protein (MIP)-1alpha, and MIP-1beta in blood MNC from patients with ischemic stroke. "
|4.||Acquired Immunodeficiency Syndrome (AIDS)
01/01/2000 - "Our results show that cocaine selectively downregulates endogenous MIP-1beta secretion by normal peripheral blood mononuclear cells (PBMC), while cocaine did not affect the MIP-1beta production by PBMC from AIDS patients. "
01/01/1998 - "CD4+ clones from nonprogressors and CD8+ clones from AIDS patients secreted high levels of RANTES, MIP1alpha, and MIP-1beta. "
12/05/2000 - "Asymptomatic HIV(+) subjects produced significantly higher levels of MIP-1alpha and MIP-1beta, but not RANTES, than uninfected donors or patients that progressed to AIDS. "
01/01/1998 - "In contrast, CD4+ clones from AIDS patients produced no RANTES and little or no MIP-1alpha or MIP-1beta. "
06/06/2008 - "The discovery that the CC chemokines RANTES, MIP-1alpha and MIP-1beta act as potent natural inhibitors of HIV-1, the causative agent of AIDS, and the subsequent identification of CCR5 as a major virus coreceptor have triggered a wealth of basic and applied research approaches aimed at developing safe and effective viral entry inhibitors. "
03/04/2010 - "Paracrine induction of cytokine secretion by co-culture was similar in normoxia and in hypoxia with IL-8 (60-80-fold induction) >IL-6 and MIP-1beta (10-20-fold) >MIP-1alpha and MCP-1 (3-10-fold). "
05/15/2007 - "Elevated levels of proinflammatory mediators IL-6, IL-8, IFN-gamma, and MIP-1beta, as well as of the regulatory cytokine IL-10, may be protective against hypoxia in bronchiolitis."
|5.||Interleukin-8 (Interleukin 8)
|6.||Chemokine CCL2 (Monocyte Chemoattractant Protein 1)
|7.||Interferon-gamma (Interferon, gamma)
|8.||Interleukin-6 (Interleukin 6)
|9.||Interleukin-10 (Interleukin 10)
|10.||Interleukin-4 (Interleukin 4)
|1.||Highly Active Antiretroviral Therapy (HAART)