|1.||Tucker, Stephen J: 1 article (05/2014)|
|2.||Veale, Emma L: 1 article (05/2014)|
|3.||Stevens, Edward B: 1 article (05/2014)|
|4.||Bajaria, Naina: 1 article (05/2014)|
|5.||Al-Moubarak, Ehab: 1 article (05/2014)|
|6.||Cao, Lishuang: 1 article (05/2014)|
|7.||Mathie, Alistair: 1 article (05/2014)|
|8.||Omoto, Kiyoyuki: 1 article (05/2014)|
|9.||Krautwurst, Dietmar: 1 article (04/2011)|
|10.||Riehle, Marc: 1 article (04/2011)|
|1.||Dysmenorrhea (Menstrual Pain)
07/01/1982 - "The fenamates appear to be effective in dysmenorrhea, although they were not studied extensively with placebo and previous experience with mefenamic acid has led to warnings about side effects. "
05/20/1988 - "Many clinical trials (controlled and uncontrolled) have demonstrated the efficacy of NSAIDs such as the fenamates, indole-acetic acid derivatives, and arylpropionic acid derivatives in relieving primary dysmenorrhea as well as IUD-induced dysmenorrhea that is also due to elevated prostaglandin levels. "
05/01/2014 - "These results provide insight into the mechanism of TREK1 channel activation by fenamates, and, given the role of TREK1 channels in pain, they suggest a novel analgesic mechanism for these compounds. "
01/01/1966 - "Effects of the fenamates on the threshold of pain."
04/01/2011 - "Fenamates are N-phenyl-substituted anthranilic acid derivatives clinically used as non-steroid anti-inflammatory drugs in pain treatment. "
01/01/1984 - "Comparisons among the fenamatic compounds, ibuprofen, indomethacin, and naproxen showed the fenamates to be more effective in providing pain relief. "
|3.||Migraine Disorders (Migraine)
01/01/1975 - "Relative anti-inflammatory potencies of aspirin, phenylbutazone, indomethacin, three fenamates and several other nonsteroidal anti-inflammatory agents were obtained in several laboratory models of acute and chronic inflammation. "
01/01/2005 - "Drugs to treat inflammation are discussed under the following headings: (1) random discoveries covering copper, salicylates, heterocyclic diones, ACTH, adrenal steroids and disease-modifying agents (DMARDs); these include Au(I)-thiolates, chloroquine, and hydroxychloroquine, minocycline, cyclosporin, salazopyrine, D-penicillamine and methotrexate; (2) programmed NSAID developments covering salicylates and fenamates, arylalkanoates, diones, non-acidic NSAIDs, clozic, lobenzarit and coxibs; (3) synthetic glucocorticosteroids; and (4) 'Biologicals' for neutralising pro-inflammatory cytokines. "
|1.||Non-Steroidal Anti-Inflammatory Agents (NSAIDs)
|2.||indoleacetic acid (indole acetic acid)
|5.||Mefenamic Acid (Pontal)
|7.||Prostaglandin Antagonists (Prostaglandin Antagonist)
|9.||Cyclooxygenase 2 Inhibitors (COX-2 Inhibitors)