|1.||Jaatinen, Risto: 2 articles (01/2007 - 10/2004)|
|2.||Laurikkala, Johanna: 2 articles (01/2007 - 10/2004)|
|3.||Thesleff, Irma: 2 articles (01/2007 - 10/2004)|
|4.||Pummila, Marja: 2 articles (01/2007 - 10/2004)|
|5.||Schneider, Pascal: 2 articles (01/2007 - 10/2004)|
|6.||Mikkola, Marja L: 2 articles (01/2007 - 10/2004)|
|7.||James, Martyn J: 1 article (01/2007)|
|8.||Fliniaux, Ingrid: 1 article (01/2007)|
|9.||Coates, Joan R: 1 article (01/2005)|
|10.||Schnabel, Robert D: 1 article (01/2005)|
|1.||Ectodermal Dysplasia (Aplasia Cutis Congenita)
01/01/2005 - "EDAR, the gene for the ectodysplasin A1 receptor, occurs on HSA2 but was excluded as the cause of canine ectodermal dysplasia."
01/26/2001 - "The ectodermal dysplasia receptor (EDAR) is a recently isolated member of the tumor necrosis factor receptor family that has been shown to play a key role in the process of ectodermal differentiation. "
10/01/2002 - "On the other hand, hypohidrotic/anhidrotic ectodermal dysplasia (HED/EDA) has been ascribed to at least three genes also involved in NF-kappaB activation: ectodysplasin (EDA1), EDA-receptor (EDAR) and EDAR-associated death domain (EDARADD). "
06/25/2002 - "TRAF6 does not associate with the cytoplasmic tail of the dl protein (DL)/ectodysplasin receptor (EDAR) receptor, which, when mutated, results in hypohidrotic (anhidrotic) ectodermal dysplasia. "
01/01/2007 - "Ectodysplasin-A (Eda), a tumour necrosis factor-like signalling molecule, and its receptor Edar are required for the development of a number of ectodermal organs in vertebrates. "
10/01/2004 - "Ectodysplasin (Eda) is a unique signalling molecule in the tumour necrosis factor family that, together with its receptor Edar, is necessary for normal development of ectodermal organs both in humans and mice. "
|3.||TNF Receptor-Associated Factor 6 (TRAF6)
|4.||Tumor Necrosis Factor Receptors (Tumor Necrosis Factor Receptor)
|5.||NF-kappa B (NF-kB)