|1.||Zhu, Huijun: 2 articles (01/2015 - 12/2014)|
|2.||Wang, Xudong: 2 articles (01/2015 - 12/2014)|
|3.||Huang, Jianfei: 2 articles (01/2015 - 12/2014)|
|4.||Saftig, Paul: 2 articles (09/2004 - 03/2003)|
|5.||Eskelinen, Eeva-Liisa: 2 articles (09/2004 - 03/2003)|
|6.||Furuta, K: 2 articles (08/2001 - 06/2001)|
|7.||Li, Xiao: 1 article (10/2015)|
|8.||Huang, J: 1 article (10/2015)|
|9.||Chen, L: 1 article (10/2015)|
|10.||Zou, Zhen: 1 article (10/2015)|
01/01/1995 - "tuberculosis stain intensely for lysosomal membrane glycoproteins and cathepsin D. "
01/01/1995 - "tuberculosis phagosome exhibits delayed clearance of major histocompatibility complex (MHC) class I molecules, relatively intense staining for MHC class II molecules and the endosomal marker transferrin receptor, and relatively weak staining for the lysosomal membrane glycoproteins, CD63, LAMP-1, and LAMP-2 and the lysosomal acid protease, cathepsin D. "
|2.||Fabry Disease (Fabry's Disease)
02/15/2014 - "This study investigated the potential relationship between the expression levels of lysosome-associated membrane proteins (LAMP) 1 and 2 and responses to enzyme replacement therapy (ERT) in the members of a single family with Fabry disease (FD). "
01/01/2002 - "The aim of this paper is to review the biology of acid hydrolases and lysosomal membrane proteins, to describe the systematic classification of LSDs and the most recently identified entities, and to briefly review novel therapeutic approaches for two lipidoses: Gaucher disease and Fabry disease."
|3.||Lysosomal Storage Diseases (Lysosomal Storage Disease)
10/01/2015 - "This study shows that the H5N1 influenza virus could induce lysosomal rupture through deglycosylating lysosome-associated membrane proteins (LAMPs) mediated by the neuraminidase activity of NA protein. "
10/01/2015 - "Neuraminidase of Influenza A Virus Binds Lysosome-Associated Membrane Proteins Directly and Induces Lysosome Rupture."
10/01/2015 - "Recently, lysosomal cell death has been defined as "lysosomal membrane permeabilization and the consequent leakage of lysosome contents into cytosol." Here, we show that the neuraminidase (NA) of H5N1 influenza A virus markedly deglycosylates and degrades lysosome-associated membrane proteins (LAMPs; the most abundant membrane proteins of lysosome), which induces lysosomal rupture, and finally leads to cell death of alveolar epithelial carcinoma A549 cells and human tracheal epithelial cells. "
10/01/2006 - "Mucopolysaccharidosis IIIC (MPS IIIC), or Sanfilippo C, represents the only MPS disorder in which the responsible gene has not been identified; however, the gene has been localized to the pericentromeric region of chromosome 8. In an ongoing proteomics study of mouse lysosomal membrane proteins, we identified an unknown protein whose human homolog, TMEM76, was encoded by a gene that maps to 8p11.1. A full-length mouse expressed sequence tag was expressed in human MPS IIIC fibroblasts, and its protein product localized to the lysosome and corrected the enzymatic defect. "
|1.||Transferrin Receptors (Transferrin Receptor)
|6.||Activating Transcription Factor 4
|7.||Galectin 1 (LGALS1)
|9.||Collagen Type I (Type I Collagen)
|1.||Enzyme Replacement Therapy