|1.||Ellard, S: 4 articles (11/2007 - 11/2000)|
|2.||Hattersley, A T: 3 articles (11/2007 - 05/2004)|
|3.||Ou, Chengshan: 2 articles (08/2015 - 01/2014)|
|4.||Wang, Yinghui: 2 articles (08/2015 - 01/2014)|
|5.||Ding, Zhenhua: 2 articles (08/2015 - 01/2014)|
|6.||Zhou, Meijuan: 2 articles (08/2015 - 01/2014)|
|7.||Liu, Hongxia: 2 articles (08/2015 - 01/2014)|
|8.||Zhou, Liang: 2 articles (08/2015 - 01/2014)|
|9.||Glubrecht, Darryl D: 2 articles (08/2013 - 08/2009)|
|10.||Godbout, Roseline: 2 articles (08/2013 - 08/2009)|
|1.||Type 2 Diabetes Mellitus (MODY)
05/01/2013 - "Mutations in the HNF1A gene encoding hepatocyte nuclear factor-1 alpha are the most common cause of MODY in most adult populations studied. "
03/01/2012 - "The patient was investigated for MODY 2 and MODY 3, and a mutation of hepatocyte nuclear factor-1 alpha gene was found. "
03/01/2011 - "The term "maturity onset diabetes of the young" (MODY) describes a heterogeneous group of monogenic diabetes of which hepatic nuclear factor-1 alpha (HNF-1α) MODY is the most common. "
01/01/2009 - "Type 2 diabetes mellitus (T2DM) is a genetically heterogeneous disease, hepatocyte nuclear factor-1 homeobox A (HNF1A) single-nucleotide polymorphisms (SNPs) playing a minor role in its pathogenesis. "
03/01/2004 - "Hepatocyte nuclear factor-1 alpha gene inactivation: cosegregation between liver adenomatosis and diabetes phenotypes in two maturity-onset diabetes of the young (MODY)3 families."
|2.||Squamous Cell Carcinoma (Epidermoid Carcinoma)
01/01/2014 - "In this study, we identified Nuclear Factor I/B (NFIB) as one of the targets of miR-365 which was previously verified as an onco-miR in cutaneous squamous cell carcinoma (CSCC). "
01/01/2014 - "miR-365 promotes cutaneous squamous cell carcinoma (CSCC) through targeting nuclear factor I/B (NFIB)."
12/01/2015 - "MiR-1290 promotes cancer progression by targeting nuclear factor I/X(NFIX) in esophageal squamous cell carcinoma (ESCC)."
08/01/2015 - "microRNA-365-targeted nuclear factor I/B transcriptionally represses cyclin-dependent kinase 6 and 4 to inhibit the progression of cutaneous squamous cell carcinoma."
08/01/2015 - "Characterizing this transcriptional repression of cyclin-dependent kinases 6 and 4 by nuclear factor I/B contributes to the understanding of the transcriptional regulation of cyclin-dependent kinases by transcription factors and also facilitates the development of new therapeutic regimens to improve the clinical treatment of cutaneous squamous cell carcinoma. "
08/01/1996 - "After adenoviral infection, nuclear factor 1 and coiled bodies were found associated with some viral replication domains. "
06/01/2002 - "Previous studies indicate that a double-nucleotide mutation that occurs frequently during chronic HBV infection converts a nuclear receptor binding site in the core promoter to the binding site of the transcription factor hepatocyte nuclear factor-1 (HNF-1) and specifically suppresses the transcription of the precore RNA. "
12/01/2003 - "Expression profiling in ovarian clear cell carcinoma: identification of hepatocyte nuclear factor-1 beta as a molecular marker and a possible molecular target for therapy of ovarian clear cell carcinoma."
11/01/2013 - "Our study used immunohistochemistry to compare the expression of estrogen receptor (ER), hepatocyte nuclear factor-1 beta (HNF1ß), p53, phosphatase and tensin homolog (PTEN), BAF250a, and cyclooxygenase-2 (COX-2) among 79 cases of endometriosis-associated ovarian cancers, including 40 (50%) clear cell carcinomas (CCCs), 33 (41%) endometrioid (EM) adenocarcinomas, four (5%) serous carcinomas, one adenosquamous carcinoma, and one adenosarcoma. "
|1.||Transcription Factors (Transcription Factor)
|3.||Cyclooxygenase 2 (Cyclooxygenase-2)
|4.||Tretinoin (Retinoic Acid)
|5.||RNA (Ribonucleic Acid)
|7.||Deoxyribonuclease I (Nickase)