|1.||Bazhan, N M: 1 article (06/2015)|
|2.||Baklanov, A V: 1 article (06/2015)|
|3.||Zhang, Xiujuan: 1 article (06/2014)|
|4.||Li, Min: 1 article (06/2014)|
|5.||Jiang, Xiuyun: 1 article (06/2014)|
|6.||Zhao, Jia Jun: 1 article (06/2014)|
|7.||Gao, Ling: 1 article (06/2014)|
|8.||Han, Wenxia: 1 article (06/2014)|
|9.||Li, Xinhua: 1 article (02/2014)|
|10.||Ferraris, Ronaldo P: 1 article (02/2014)|
12/21/1992 - "Western blot analysis showed that the amount of GLUT2 protein in BLM vesicles was increased in rats with diabetes and those given a high-carbohydrate diet, but not in those with experimental hyperglycemia. "
01/01/1995 - "Densitometric analysis of Western blots on graft homogenates demonstrated that GLUT2 protein levels in the islets, when exposed to chronic hyperglycemia for 2 weeks, were decreased to 16% of those of normal recipients. "
01/01/1998 - "Significant reduction of both GLUT2 protein and mRNA expression was first noted 1 day after the third STZ injection, clearly preceding the onset of hyperglycemia. "
05/01/2007 - "Therefore, the effects of HJ on STZ-induced hyperglycemia in rats could be summarized as follows: besides increasing insulin synthesis and release, HJ normalizes GLUT2 protein expression in the liver to suppress hyperglycemia. "
02/01/1997 - "These data suggest that 1) the loss of GLUT2 protein associated with hyperglycemia is at least partially explained by reduced levels of the GLUT2 gene transcripts; 2) the reduction of beta-cell insulin content during chronic hyperglycemia may not be completely due to degranulation (reduced levels of gene transcripts may play a role); and 3) the reduction in the transcription factor IDX-1 raises the possibility that dysregulation of transcription factors may contribute to the abnormal beta-cell function found in states of chronic hyperglycemia."
|3.||Pituitary Neoplasms (Pituitary Adenoma)
12/01/1998 - "We investigated the mechanisms of insulin secretion by transfecting into a pituitary adenoma cell line (AtT20) a combination of genes encoding human insulin (HI), glucose transporter type 2 (GLUT2) and glucokinase (GK), followed by studying the characteristics of these cells. "
12/01/1998 - "Cellular characterization of pituitary adenoma cell line (AtT20 cell) transfected with insulin, glucose transporter type 2 (GLUT2) and glucokinase genes: insulin secretion in response to physiological concentrations of glucose."
06/01/2015 - "The relative gene expressions of glucose-6-phosphatase (G6P), phosphoenolpyruvate carbo- xykinase (PEPCK)--markers of gluconeogenesis, glucokinase (GK)--a marker of glycolysis, glucose transporter type 2 (GLUT2)--a marker of input and output of glucose in the liver were measured during the development of melanocortin (MC) obesity in male mice of C57BL/6J strain with mutation yellow in the Agouti locus (Ay/a mice). "
|1.||Messenger RNA (mRNA)
|5.||Facilitative Glucose Transport Proteins (Glucose Transporter)
|7.||Transcription Factors (Transcription Factor)
|10.||Glucose-6-Phosphatase (Glucose 6 Phosphatase)