|1.||Lisanti, Michael P: 12 articles (01/2015 - 01/2002)|
|2.||Lisanti, M P: 7 articles (02/2004 - 03/2000)|
|3.||Ishikawa, Yoshihiro: 6 articles (04/2014 - 12/2002)|
|4.||Sunada, Yoshihide: 6 articles (01/2013 - 01/2004)|
|5.||Minetti, C: 6 articles (01/2011 - 03/2000)|
|6.||Roth, David M: 5 articles (09/2015 - 01/2008)|
|7.||Patel, Hemal H: 5 articles (09/2015 - 01/2008)|
|8.||Balijepalli, Ravi C: 5 articles (09/2015 - 11/2006)|
|9.||Tsutsumi, Yasuo M: 5 articles (04/2014 - 01/2008)|
|10.||Ohsawa, Yutaka: 5 articles (01/2013 - 01/2004)|
08/01/2015 - "Inhibition of PKCβ2 overexpression ameliorates myocardial ischaemia/reperfusion injury in diabetic rats via restoring caveolin-3/Akt signaling."
01/01/2008 - "We conclude that caveolae and caveolin-3 are critical for volatile anesthetic-induced protection of the heart from ischemia/reperfusion injury."
04/01/2012 - "Compared with wild-type mice, caveolin-3 knockout (Cav-3KO) mice exhibited modestly increased myocardial ischemia/reperfusion injury (increased infarct size, apoptosis, and poorer cardiac function recovery; P<0.05). "
01/01/2014 - "We conclude that caveolae and caveolin-3 are critical for exendin-4 induced protection of the heart from ischemia/reperfusion injury."
05/01/2010 - "An in vivo mouse model of ischemia-reperfusion injury with delayed anesthetic preconditioning (APC) was tested in wild-type, caveolin-1 knockout, and caveolin-3 knockout mice. "
02/01/2009 - "Loss of caveolin-3 induces a lactogenic microenvironment that is protective against mammary tumor formation."
01/01/2002 - "Samples with high levels of caveolin-3 tended to have higher degrees of tumor-infiltrating immune cells, although this finding was not statistically significant. "
01/01/2002 - "Tumor-infiltrating immune cells were also studied immunohistochemically, and the correlation between the number of immune cells and caveolin-3 concentrations was calculated. "
01/01/2015 - "The purpose of this study was to investigate whether MURC/cavin-4, a plasma membrane and Z-line associated protein exhibiting an overlapping distribution with Caveolin-3 (Cav-3) in heart and muscle tissues, may be expressed and play a role in rhabdomyosarcoma (RMS), an aggressive myogenic tumor affecting childhood. "
01/01/2015 - "MURC/cavin-4 Is Co-Expressed with Caveolin-3 in Rhabdomyosarcoma Tumors and Its Silencing Prevents Myogenic Differentiation in the Human Embryonal RD Cell Line."
|3.||Cardiomegaly (Heart Hypertrophy)
05/31/2011 - "We hypothesized that cardiac myocyte-specific overexpression of caveolin-3 (Cav-3), a muscle-specific caveolin, would alter natriuretic peptide signaling and attenuate cardiac hypertrophy. "
05/31/2011 - "Cardiac-specific overexpression of caveolin-3 attenuates cardiac hypertrophy and increases natriuretic peptide expression and signaling."
04/30/2009 - "Our data suggest that the alterations in nuclear and mitochondrial activation of STAT3 and caveolae localization of caveolin-3 are related to the development of the catecholamine-induced cardiac hypertrophy."
04/30/2009 - "Modulation of the caveolin-3 localization to caveolae and STAT3 to mitochondria by catecholamine-induced cardiac hypertrophy in H9c2 cardiomyoblasts."
09/01/2001 - "A decrease in caveolin-3 expression may play a role in the development of cardiac hypertrophy in SHR, presumably through de-regulating the inhibition of growth signal in the hearts of SHR in the hypertrophic stage."
11/01/2003 - "Our results indicate that overexpression of caveolin-3 causes severe cardiac tissue degeneration, fibrosis and a reduction in cardiac functions. "
11/01/2015 - "Bin1 loss increased interstitial fibrosis and mislocalization of the voltage-dependent calcium channel Cav 1.2, and the lipid raft scaffold protein caveolin-3, which normally complexes with Bin1 and Cav 1.2 in cardiomyocyte membranes. "
03/01/2013 - "In contrast, transgenic overexpression of profilin-1 in SHRs induced more serious cardiac hypertrophy and fibrosis with significant reduction of sarcolemmal caveolae, caveolin-3 protein, eNOS activity, and production of NO when compared with SHR controls. "
03/01/2013 - "Lowering profilin-1 expression in SHRs significantly attenuated hypertension-induced cardiac hypertrophy and fibrosis and displayed a significant preservation of myofibrils, sarcolemmal caveolae, abundance of caveolin-3 protein, activity of eNOS and production of nitric oxide (NO). "
|5.||Limb-Girdle Muscular Dystrophies (Limb-Girdle Muscular Dystrophy)
01/15/2004 - "The current study characterized the biochemical and cardiac parameters of P104L mutant caveolin-3 transgenic mice, a model of an autosomal dominant limb-girdle muscular dystrophy (LGMD1C). "
08/01/2012 - "Skeletal muscle expressing Pro104Leu mutant caveolin 3 (CAV3(P104L)) in mouse becomes atrophied and serves as a model of autosomal dominant limb-girdle muscular dystrophy 1C. "
08/01/2011 - "Mutations in the caveolin-3 gene cause autosomal dominant limb-girdle muscular dystrophy 1C (LGMD1C). "
05/01/2007 - "A deficiency in caveolin-3 (Cav-3), the major striated muscle isoform, is responsible for skeletal muscle disorders, such as limb-girdle muscular dystrophy 1C (LGMD 1C). "
11/01/2006 - "Dominant-negative mutations of the caveolin-3 gene cause autosomal dominant limb-girdle muscular dystrophy 1C (LGMD1C) with loss of caveolin-3. "
|3.||Phosphatidylinositol 3-Kinase (1 Phosphatidylinositol 3 Kinase)
|7.||alpha-Linolenic Acid (Linolenic Acid)
|8.||Factor IX (PTC)
|9.||DNA (Deoxyribonucleic Acid)
|10.||papillary Thyroid cancer