|1.||Dörken, Bernd: 6 articles (04/2006 - 06/2002)|
|2.||Daniel, Peter T: 5 articles (04/2006 - 06/2002)|
|3.||Zhang, Y: 4 articles (06/2015 - 07/2013)|
|4.||Wu, Chung-Hsin: 4 articles (01/2015 - 01/2015)|
|5.||Lin, Ching-Lung: 4 articles (01/2015 - 01/2015)|
|6.||Hsu, Chih-Hsiang: 4 articles (01/2015 - 01/2015)|
|7.||Sheu, Shuenn-Jyi: 4 articles (01/2015 - 01/2015)|
|8.||Wang, Sheue-Er: 4 articles (01/2015 - 01/2015)|
|9.||Sturm, Isrid: 4 articles (04/2006 - 06/2002)|
|10.||Wang, Li: 3 articles (12/2015 - 12/2008)|
01/01/2007 - "The results of the study demonstrated the following 1) immediate in vivo activation of endogenous proapoptotic Bax protein in disseminated tumor cells, not protected by Bcl-2 against apoptosis, and its correlation with accelerated tumor progression; 2) complete suppression of in vivo Bax activation in tumor cells protected by Bcl-expression, and inhibited tumor progression; 3) alternative character of Bcl-2 and Bax expression under the conditions of accelerated and inhibited tumor progression. "
09/29/2015 - "Inhibition of VTRNA2-1-5p increases Bax protein expression and apoptotic cell death in cervical cancer cells. "
01/01/2015 - "For multiple comparisons, ANOVA (differences in tumor volumes, growth rates, apoptosis, proliferation indexes, and Bcl-2 and Bax protein levels) was used when the data were normally distributed with homogenous variance, and rank sum tests were used otherwise. "
04/15/2014 - "We demonstrate that mitochondria-associated Bcl2L13 inhibits apoptosis induced by a wide spectrum of chemo- and targeted therapies upstream of Bcl2-associated X protein activation and mitochondrial outer membrane permeabilization in vitro and promotes GBM tumor growth in vivo. "
04/01/2013 - "High levels of Bcl-2-associated X protein (Bax) in tumor cells have been found to promote apoptosis and sensitize cells to anti-cancer therapies. "
01/15/2006 - "These results suggest that the protective effects of DNP against I/R injury would be mediated, at least in part, through the increased ratio of Bcl-2 to Bax protein after ischemia-reperfusion."
01/01/2015 - "Protein levels of Akt, phosphorylated (activated) Akt (P-Akt), as well as contents of BAX protein were assayed (Western blotting) in cytosolic fraction of myocardial tissue samples taken prior to and after 30-min global ischemia and 40-min reperfusion. "
01/01/2002 - "Moreover, it was demonstrated that the kinetics of induced p53, p21(WAF-1) and Bax protein differ between 3 hours and 6 hours of ischemia, and it is speculated that this difference plays an important role in determining the consequence of the cell exposed to ischemia."
01/01/2002 - "The authors investigated the expression of p53, p21(WAF-1), Bax protein, and apoptosis to elucidate the cellular response to ischemia-reperfusion of skeletal muscle using the rat lower limb model. "
01/01/2002 - "Difference of molecular response to ischemia-reperfusion of rat skeletal muscle as a function of ischemic time: study of the expression of p53, p21(WAF-1), Bax protein, and apoptosis."
|3.||B-Cell Lymphoma (Lymphoma, B Cell)
06/01/2014 - "It was also demonstrated that Met promoted A498 cell apoptosis and mechanistic studies suggested that this was mediated by the downregulation of B-cell lymphoma 2 and concurrent upregulation of Bcl-2-associated X protein. "
08/01/2015 - "The expression of B-cell lymphoma 2 (Bcl-2), Bcl-2-associated X protein (Bax), phosphorylated (p)-ERK1/2, p-JNK and p-p38 were also examined using western blot analysis. "
03/01/2015 - "The decrease of anti‑apoptotic B-cell lymphoma 2 (Bcl‑2) and Bcl-2 extra large protein expression was accompanied by the increase in pro‑apoptotic Bcl-2-associated X protein and P53-upregulated modulator of apoptosis expression. "
02/01/2015 - "Mutant follicles exhibited reduced levels of apoptosis, and both B-cell lymphoma 2 (Bcl-2) and BCL-2-associated X protein (Bax) were altered compared with Controls. "
10/15/2014 - "Interestingly, TCBQ has no effect on caspase family genes or B-cell lymphoma 2 (Bcl-2)/Bcl-2 associated X (Bax) protein expressions, which implied that TCBQ-induced hepatotoxicity is independent of apoptosis. "
09/01/2014 - "An upregulation of Bax protein expression and a downregulation of Bcl‑xl in a dose‑dependent manner in human leukemia K562 cells was observed. "
03/01/2007 - "In addition, Molt-4 cells treated with As2O3 exhibited the down-regulation of Bax protein expression, suggesting that Bax may be involved in accumulating of Beclin-1 and triggering autophagic cell death in As2O3-treated leukemia cells. "
01/01/2015 - "Regarding the underlying mechanisms of inducing apoptosis in DBTRG-05MG GBM cells, CCF-NLs were shown to downregulate the expression of antiapoptotic B-cell lymphoma/leukemia 2 (Bcl-2), an apoptosis-related protein family member, but the expression of proapoptotic Bcl-2-associated X protein was enhanced compared with that in controls. "
01/01/2012 - "The expression of HSP70, programmed cell death protein 4 (PDCD4), B-cell lymphoma/leukemia-2 (Bcl-2) and Bcl-2-associated X protein (Bax) was detected by Western blot. "
01/01/2014 - "Moreover, LTE fraction increased the ratio of pro-apoptotic Bcl-2-associated X protein (Bax)/anti-apoptotic myeloid cell leukemia 1 (Mcl-1) that caused mitochondrial membrane potential loss and cytochrome c released to cytosol. "
01/01/2014 - "Immunohistochemical analyses of the cytoprotective and anti-apoptotic heat-shock protein-70 (HSP-70) and the apoptotic Bax protein were performed. "
01/01/2013 - "The compound up regulated heat shock protein-70 (HSP-70) and down regulated Bax protein expression in the ulcerated tissue. "
12/08/1997 - "No change in bax protein was observed in control cells up to 8 h post heat shock. "
12/08/1997 - "bax protein levels in bcl-xL-overexpressing cells were decreased approximately 80% below baseline levels 1 h post heat shock. "
12/01/2015 - "However, gathered MNPs combined with ELFF treatment prolonged mice survival time and inhibited loading tumor cells proliferation significantly compared to other mice groups (p < 0.05); furthermore, the tumor cells early apoptosis ratio of mice group was significantly higher than other groups (p < 0.05), and ELFF combined with gathered MNPs treatment improved tumor cells early apoptosis associated with Bcl group protein expression: Bax protein expression was higher than Bcl-2 and the combined treatment improved cells Heat shock protein-27 (Hsp-27) expression which could protect cells avoiding early apoptosis. "
|1.||Caspase 3 (Caspase-3)
|2.||Messenger RNA (mRNA)
|3.||Proteins (Proteins, Gene)
|4.||Cytochromes c (Cytochrome c)
|5.||DNA (Deoxyribonucleic Acid)
|8.||Complementary DNA (cDNA)
|9.||Transferrin (beta 2 Transferrin)
|1.||Drug Therapy (Chemotherapy)