|1.||Staron, Krzysztof: 2 articles (05/2015 - 01/2014)|
|2.||Trzcinska-Danielewicz, Joanna: 2 articles (05/2015 - 01/2014)|
|3.||Orzechowska, Emilia Joanna: 2 articles (05/2015 - 01/2014)|
|4.||Girstun, Agnieszka: 1 article (05/2015)|
|5.||Faubel, Sarah: 1 article (02/2015)|
|6.||Lee, Dong Won: 1 article (02/2015)|
|7.||Edelstein, Charles L: 1 article (02/2015)|
|8.||El-Attar, R: 1 article (11/2014)|
|9.||Kulms, D: 1 article (11/2014)|
|10.||Odell, E: 1 article (11/2014)|
08/01/2010 - "Deficiency of Bid protein reduces sepsis-induced apoptosis and inflammation, while improving septic survival."
01/01/2014 - "Sitagliptin also decreased IL-1β and TNF-α levels, as well as, prevented the increase of BAX/Bcl-2 ratio, Bid protein levels, and TUNEL-positive cells which indicates protective effects against inflammation and proapoptotic state in the kidney of diabetic rats, respectively. "
01/01/2014 - "Controlled delivery of BID protein fused with TAT peptide sensitizes cancer cells to apoptosis."
05/01/2015 - "Overexpression of the BH3-interacting domain death agonist (BID) protein sensitizes certain cancer cell lines to apoptosis induced by anticancer agents, particularly by those acting through death receptors (e.g. "
05/01/2005 - "Sensitivity to anti-Fas depends on Fas, caspase-8, and Bid protein levels in cervical cancer cells. "
09/01/2013 - "Moreover, Mith also inhibited the phosphorylation of mTOR on serine 2448 and Mcl-1, and increased tBid protein in prostate tumors in athymic nude mice bearing DU145 cells as xenografts. "
11/07/2014 - "Gli1 protein regulates the S-phase checkpoint in tumor cells via Bid protein, and its inhibition sensitizes to DNA topoisomerase 1 inhibitors."
02/01/2015 - "In summary, caspase inhibition decreases caspase-1, IL-1α, and IL-1β but not NLRP3 or BID protein and protects against necrosis in cisplatin-treated freshly isolated PTs."
11/11/2014 - "Tumour necrosis factor-related apoptosis-inducing ligand sensitivity was associated with high caspase-8 and Bid protein levels, and TRAIL-sensitive cell lines were killed via the type II extrinsic apoptotic pathway. "
|5.||Hashimoto Disease (Thyroiditis, Hashimoto)
|2.||Caspase 8 (Caspase-8)
|3.||Serine Proteases (Serine Protease)
|4.||Death Domain Receptors
|1.||Heterologous Transplantation (Xenotransplantation)