|1.||Ben-Hail, Danya: 2 articles (12/2015 - 10/2015)|
|2.||Shoshan-Barmatz, Varda: 2 articles (12/2015 - 10/2015)|
|3.||Reddy, P Hemachandra: 2 articles (12/2013 - 12/2012)|
|4.||Manczak, Maria: 2 articles (12/2013 - 12/2012)|
|5.||Mizrachi, Dario: 1 article (12/2015)|
|6.||Dangoor, Liron: 1 article (12/2015)|
|7.||Smilansky, Angela: 1 article (12/2015)|
|8.||Nakdimon, Itay: 1 article (12/2015)|
|9.||Gao, Zongwei: 1 article (11/2015)|
|10.||Shang, Qingjuan: 1 article (11/2015)|
|1.||Alzheimer Disease (Alzheimer's Disease)
12/01/2012 - "The purpose of our study was to determine the relationship between voltage-dependent anion channel 1 protein (VDAC1) and amyloid beta (Aβ) and phosphorylated tau in Alzheimer's disease (AD). "
12/25/2015 - "The Voltage-dependent Anion Channel 1 Mediates Amyloid β Toxicity and Represents a Potential Target for Alzheimer Disease Therapy."
05/01/2013 - "Additionally, the mitochondrial voltage-dependent anion channel 1 (VDAC1), which is involved in the release of apoptotic proteins with possible relevance in Alzheimer's disease (AD) neuropathology, plays an important role in maintaining mitochondrial function and integrity. "
01/01/2011 - "The mitochondrial voltage-dependent anion channel 1 (VDAC1) is involved in the release of apoptotic proteins with possible relevance in Alzheimer's disease (AD) neuropathology. "
01/01/2011 - "Enhanced expression of the voltage-dependent anion channel 1 (VDAC1) in Alzheimer's disease transgenic mice: an insight into the pathogenic effects of amyloid-β."
10/01/2015 - "The mitochondrial voltage-dependent anion channel 1 in tumor cells."
12/01/2008 - "The authors recently demonstrated that the mitochondrial voltage-dependent anion channel 1 (VDAC1) is involved in the sensitivity of cancer cells to furanonaphthoquinone (FNQ). "
05/01/2011 - "The rupture of HK:VDAC1 protein complex provides a therapeutic opportunity, as this association appears to protect tumor cells from mitochondrial outer membrane permeabilization, an event that marks the point of no return in multiple pathways leading to cell death. "
05/01/2011 - "Hexokinase, the first enzyme in the glycolytic pathway, not only improves the cell's energy supply in malignant cells, but also protects cancer cells against apoptosis through direct interaction with mitochondria and with the Voltage Dependent Anion Channel 1 (VDAC1). "
04/01/2011 - "Changes in protein levels were validated by immunoprobing 1D blots, confirming increases in heat shock cognate 71 kDa (Hsc70), 60 kDa heat shock protein (Hsp60), fumarase, glutamate oxaloacetate transaminase 2, ATP synthase subunit d, and voltage-dependent anion-channel 1 (VDAC1). "
10/01/2001 - "Northern blotting confirmed increased expression of skeletal muscle alpha-actin (ACTA1), myosin light chain 2a (MLC2a), GTP-binding protein Gs-alpha subunit (GNAS1), NADH ubiquinone oxidoreductase (NDUFB10), voltage-dependent anion channel 1 (VDAC1), four-and-a-half LIM domain protein 1 (FHL1) (also known as SLIM1), sarcosin (SARCOSIN) and heat shock 70kD protein 8 (HSPA8) by less than twofold. "
|5.||Stomach Neoplasms (Stomach Cancer)
|1.||Amyloid (Amyloid Fibrils)
|2.||GTP-Binding Proteins (G-Protein)
|3.||Proteins (Proteins, Gene)
|4.||Carrier Proteins (Binding Protein)
|5.||AMP-Activated Protein Kinases
|8.||Casein Kinase II (Casein Kinase 2)
|9.||Long-Chain Acyl-CoA Dehydrogenase (Long-Chain-Acyl-Coenzyme A Dehydrogenase)
|10.||Electron Transport Complex I (NADH-CoQ Reductase)