|1.||Rachakonda, P Sivaramakrishna: 2 articles (02/2014 - 10/2013)|
|2.||Hemminki, Kari: 2 articles (02/2014 - 10/2013)|
|3.||Heidenreich, Barbara: 2 articles (02/2014 - 10/2013)|
|4.||Kumar, Rajiv: 2 articles (02/2014 - 10/2013)|
|5.||Thiel, Gerald: 2 articles (03/2011 - 08/2010)|
|6.||Endo, Takeshi: 2 articles (03/2011 - 08/2010)|
|7.||Müller, Isabelle: 2 articles (03/2011 - 08/2010)|
|8.||Wiklund, N Peter: 1 article (10/2013)|
|9.||Hosen, Ismail: 1 article (10/2013)|
|10.||Ryk, Charlotta: 1 article (10/2013)|
|1.||Breast Neoplasms (Breast Cancer)
03/15/2001 - "Interestingly, our studies reveal that although both Elk-1 and SAP-1 are highly homologous members of a subfamily of ETS domain proteins called ternary complex factors, it is only Elk-1 but not SAP-1 that can augment the growth suppressive function of BRCA1a/1b proteins in breast cancer cells. "
10/22/2013 - "The somatic mutations, mainly at positions -124 and -146 bp from ATG start site that create binding motifs for E-twenty six/ternary complex factors (Ets/TCF), affected 65.4% of the tumors, with even distribution across different stages and grades. "
02/01/2014 - "The newly described germline and recurrent somatic mutations in melanoma and other cancers in the TERT promoter that create de novo E-twenty six/ternary complex factors (Ets/TCF) binding sites, provide an insight into the possible cause of tumor-specific increased TERT expression. "
09/01/2002 - "Here we address the question of how much activation of transformation-relevant transcription factors [activator protein-1 (AP-1), ternary complex factors (TCFs), or nuclear factor kappa-B (NF-kappa-B)] is required for transformation response and how much tumor promoter produces significant risk of transformation. "
03/25/2011 - "Signal transduction of pregnenolone sulfate in insulinoma cells: activation of Egr-1 expression involving TRPM3, voltage-gated calcium channels, ERK, and ternary complex factors."
08/15/2010 - "Expression of dominant-negative mutants of c-Jun and Elk-1 reduced AP-1 transcriptional activity in INS-1 cells indicating that c-Jun and ternary complex factors are involved in the regulation of AP-1 activity in glucose-stimulated insulinoma cells."
|4.||T-Cell Leukemia (Leukemia, T Cell)
|5.||Melanoma (Melanoma, Malignant)
|1.||Proteins (Proteins, Gene)
|2.||Transcription Factor AP-1 (Transcription Factor AP 1)
|3.||NF-kappa B (NF-kB)
|4.||Calcium Channels (Calcium Channel)
|5.||Transcription Factors (Transcription Factor)