|1.||Loffing, Johannes: 4 articles (12/2015 - 08/2008)|
|2.||Devuyst, Olivier: 2 articles (09/2014 - 03/2014)|
|3.||Hoorn, Ewout J: 2 articles (09/2012 - 10/2011)|
|4.||Ellison, David H: 2 articles (11/2011 - 10/2011)|
|5.||Yang, Chao-Ling: 2 articles (11/2011 - 10/2011)|
|6.||McCormick, James A: 2 articles (11/2011 - 10/2011)|
|7.||Hsu, Yu-Juei: 2 articles (04/2009 - 02/2004)|
|8.||Lin, Shih-Hua: 2 articles (04/2009 - 02/2004)|
|9.||Wainford, Richard D: 1 article (01/2016)|
|10.||Kuwabara, Jill T: 1 article (01/2016)|
06/01/2003 - "To demonstrate the long-term efficacy of intranasal furosemide, an inhibitor of the sodium chloride cotransporter channel at the basolateral surface of the respiratory epithelial cell, vs no therapeutic intervention vs intranasal mometasone furoate, a corticosteroid, in preventing relapses of chronic hyperplastic sinusitis with nasal polyposis. "
|2.||Hyperaldosteronism (Conn Syndrome)
09/01/2012 - "The phosphorylated sodium chloride cotransporter in urinary exosomes is superior to prostasin as a marker for aldosteronism."
09/01/2012 - "Here, we hypothesized that the phosphorylated (active) form of the sodium chloride cotransporter (pNCC) or prostasin could serve as biomarkers for aldosteronism. "
|3.||Hypertension (High Blood Pressure)
03/01/2008 - "Studies using transgenic animals and gene knockins have clarified the role of mutant WNK4 in hypertension, by revealing its main action to be increasing the expression and activity of sodium-chloride cotransporter (NCC) in the kidney. "
11/01/2011 - "Overexpression of the sodium chloride cotransporter is not sufficient to cause familial hyperkalemic hypertension."
02/01/2011 - "Hypertension, dietary salt intake, and the role of the thiazide-sensitive sodium chloride transporter NCCT."
01/01/2009 - "The gene which encodes for the thiazide- sensitive sodium-chloride cotransporter (NCCT) represents a possible link between hypertension and osteoporosis. "
01/15/2016 - "Recent studies have implicated a role of norepinephrine (NE) in the activation of the sodium chloride cotransporter (NCC) to drive the development of salt-sensitive hypertension. "
09/01/2015 - "CR = clearance rate ΔFE = net increase in fractional excretion eGFR = estimated glomerular filtration rate FE = fractional excretion GS = Gitelman syndrome NCC = sodium-chloride cotransporter ROC = receiver operating characteristic TRPM6 = transient receptor potential channel melastatin subtype 6."
02/01/2015 - "A variety of genetic backgrounds cause the loss of function of thiazide-sensitive sodium chloride cotransporter, encoded by SLC12A3, responsible for the phenotypes in Gitelman syndrome. "
09/01/2014 - "Genetic analysis evidenced a homozygous mutation (p.Arg399Cys) in the SLC12A3 gene coding for the sodium-chloride cotransporter (NCC), confirming the diagnosis of Gitelman syndrome. "
01/01/2014 - "A presumptive diagnosis of Gitelman syndrome (GS) was then made and the patient's peripheral blood mononuclear cells were subjected to sequence analysis of the SLC12A3 gene, which encodes a thiazide-sensitive sodium-chloride cotransporter. "
01/01/2013 - "Gitelman syndrome (GS) is a renal tubular disorder of the thiazide-sensitive sodium chloride cotransporter, which is located in the distal tubule of the loop of Henle. "
03/01/2012 - "Gitelman's syndrome is an inherited tubulopathy affecting thiazide-sensitive sodium chloride cotransporter, which manifests with hypokalemic alkalosis, hypomagnesemia, and hypocalciuria. "
04/01/2009 - "Gitelman's syndrome is a hereditary disorder occurring due to loss of functional mutations of the gene encoding the distal convoluted tubule sodium chloride cotransporter (NCCT) and is characterized by hypokalemic metabolic alkalosis, hypomagnesemia and hypocalciuria. "
01/01/2010 - "Gitelman syndrome and thiazide diuretics which inhibit sodium chloride cotransporter in the distal convoluted tubule are associated with hypokalemia, metabolic alkalosis, renal magnesium wasting, hypomagnesemia, and hypocalciuria. "
|3.||mometasone furoate (mometasone)
|5.||Sodium Chloride Symporter Inhibitors (Diuretics, Thiazide)
|6.||Mineralocorticoid Receptors (Mineralocorticoid Receptor)
|7.||Sodium Channels (Sodium Channel)
|1.||Transplantation (Transplant Recipients)