|1.||Zeng, Jibin: 1 article (12/2003)|
|2.||Hassan, A Jacob: 1 article (12/2003)|
|3.||Morales, Carlos R: 1 article (12/2003)|
|4.||Lefrancois, Stephane: 1 article (12/2003)|
|5.||Canuel, Maryssa: 1 article (12/2003)|
|6.||Suzuki, Kunihiko: 1 article (09/2003)|
|7.||Whitelegge, J P: 1 article (07/2003)|
|8.||Sung, H: 1 article (07/2003)|
|9.||Fluharty, C B: 1 article (07/2003)|
|10.||Stevens, R L: 1 article (07/2003)|
|1.||Neuronal Ceroid-Lipofuscinoses (Neuronal Ceroid Lipofuscinosis)
01/01/1995 - "Immunological studies on sphingolipid activator proteins in the neuronal ceroid-lipofuscinoses."
01/01/1995 - "Sphingolipid activator proteins in the neuronal ceroid-lipofuscinoses: an immunological study."
02/01/1997 - "Sphingolipid activator proteins (SAPs) in neuronal ceroid lipofuscinoses (NCL)."
02/01/1997 - "Based on the predominant component of the storage material the neuronal ceroid lipofuscinoses (NCL) can be divided into two categories: one storing mitochondrial ATP synthase subunit c and the other storing sphingolipid activator proteins (SAPs). "
11/01/1995 - "The nosologic significance of both the subunit C of the adenosine triphosphate synthase and the sphingolipid activator proteins, although they make up a considerable amount of the crude auto-fluorescent lipopigments in neuronal ceroid-lipofuscinosis, is still unclear. "
08/01/1998 - "The inherited deficiencies of lysosomal hydrolases and of sphingolipid activator proteins both give rise to sphingolipid storage diseases. "
10/09/1997 - "Both, the inherited deficiency of lysosomal hydrolases and of sphingolipid activator proteins give rise to sphingolipid storage diseases. "
03/01/1996 - "The inherited deficiency of both lysosomal hydrolases and sphingolipid activator proteins gives rise to sphingolipid storage diseases. "
|3.||Lysosomal Storage Diseases (Lysosomal Storage Disease)
01/01/1992 - "Distribution of saposins (sphingolipid activator proteins) in tissues of lysosomal storage disease patients."
10/01/1996 - "Sphingolipid activator proteins (saposins) stimulate the degradation of glycosphingolipids by lysosomal enzymes, and defects in saposins cause accumulation of substrate lipids in the affected tissues in lysosomal storage disease. "
|4.||Neurodegenerative Diseases (Neurodegenerative Disease)
12/15/2003 - "However, the lysosomes of I-cell disease (ICD) patients, a condition resulting from a mutation in the phosphotransferase that adds mannose 6-phosphate to hydrolases, have near normal levels of several lysosomal proteins, including the sphingolipid activator proteins (SAPs), GM2AP and prosaposin. "
|4.||Mitochondrial Proton-Translocating ATPases (Mitochondrial ATP Synthase)
|9.||Adenosine Triphosphate (ATP)